Clonogenic versus morphogenic mutations in myeloid neoplasms: chronologic observations in a U2AF1, TET2, CSF3R and JAK2 ‘co-mutated’ myeloproliferative neoplasm suggest a hierarchical order of mutations and potential predictive value for kinase inhibitor treatment response

Magdalena M. GerlachInstitute of Pathology, University Hospital Basel, Basel, Switzerland;

Pontus LundbergDepartment for Hematology, University Hospital Basel, Basel, Switzerland;

Joerg HalterDepartment for Hematology, University Hospital Basel, Basel, Switzerland;

Christian ArrantoDepartment for Hematology, University Hospital Basel, Basel, Switzerland;

Friedel WenzelInstitute of Medical Genetics, University Hospital Basel, Basel, Switzerland

Stefan DirnhoferInstitute of Pathology, University Hospital Basel, Basel, Switzerland;

Alexandar TzankovInstitute of Pathology, University Hospital Basel, Basel, Switzerland; Correspondence[email protected]

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Disclosure forms provided by the authors are available with the full text of this article online at https://doi.org/10.1080/10428194.2017.1406087.

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Clonogenic versus morphogenic mutations in myeloid neoplasms: chronologic observations in a U2AF1, TET2, CSF3R and JAK2 ‘co-mutated’ myeloproliferative neoplasm suggest a hierarchical order of mutations and potential predictive value for kinase inhibitor treatment response

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Clonogenic versus morphogenic mutations in myeloid neoplasms: chronologic observations in a U2AF1, TET2, CSF3R and JAK2 ‘co-mutated’ myeloproliferative neoplasm suggest a hierarchical order of mutations and potential predictive value for kinase inhibitor treatment response

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