Octasomy 21 in a patient with secondary AML after CMML: the role of acquired NRAS mutations in triggering aneuploidy

Kathrin ThomayDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Yvonne Lisa BehrensDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Jana LentesDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Nicole WittnerDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Verena WittigDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Doreen RotheDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Doris SteinemannDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Brigitte SchlegelbergerDepartment of Human Genetics, Hannover Medical School, Hannover, Germany

Gudrun GöhringDepartment of Human Genetics, Hannover Medical School, Hannover, GermanyCorrespondence[email protected]

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Acknowledgements

We would like to thank the patient and attending doctors for their participation in this study.

This work is supported by funding from the Deutsche Forschungsgemeinschaft (DFG, German Research Foundation) for the Cluster of Excellence REBIRTH (from Regenerative Biology to Reconstructive Therapy) (EXC 62/3).

Potential conflict of interest

Disclosure forms provided by the authors are available with the full text of this article online at https://doi.org/10.1080/10428194.2018.1427861.

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Octasomy 21 in a patient with secondary AML after CMML: the role of acquired NRAS mutations in triggering aneuploidy

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Octasomy 21 in a patient with secondary AML after CMML: the role of acquired NRAS mutations in triggering aneuploidy

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