Activating STAT5B mutations can cause both primary hypereosinophilia and lymphocyte-variant hypereosinophilia

Kavita Umraua Department of Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA

Ken Naganumab Department of Hematology, Saitama Medical Center, Saitama Medical University, Saitama, Japan

Qi Gaoa Department of Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA

Ahmet Dogana Department of Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA

Masahiro Kizakib Department of Hematology, Saitama Medical Center, Saitama Medical University, Saitama, Japan

Mikhail Roshala Department of Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA

Ying Liua Department of Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA

Mariko Yabea Department of Pathology and Laboratory Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USACorrespondence[email protected]

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Disclosure statement

AD has received personal consultancy fees from InCycle, EUSA and Loxo, and has received research support from Roche and Takeda. MR participates in a scientific advisory board for and has equity in Auron Therapeutics, Inc., has received research support from Roshe, NGM and Beat AML. MY has received personal consultancy fee from Janssen Research and Development. The remaining authors declare no financial interests. No potential conflict of interest was reported by the author(s).

Activating STAT5B mutations can cause both primary hypereosinophilia and lymphocyte-variant hypereosinophilia

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Activating STAT5B mutations can cause both primary hypereosinophilia and lymphocyte-variant hypereosinophilia

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