Anti-cyclic citrullinated glucose-6-phosphate isomerase peptide-7 (CCG-7) antibodies were suppressed by biologics treatment and deposited to citrullinated proteins in CD68-positive cells in the RA synovium

Naoto UmedaDepartment of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan; ;Department of Rheumatology, Tsuchiura Kyodo General Hospital, Tsuchiura, Japan;

Isao MatsumotoDepartment of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan; Correspondence[email protected]

Yuki TanakaDepartment of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan;

Hoshimi KawaguchiDepartment of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan;

Hiroshi EbeDepartment of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan;

Yayoi KagamiMolecular Regulation of Aging, Tokyo Metropolitan Institute of Gerontology, Tokyo, Japan

Akihito IshigamiMolecular Regulation of Aging, Tokyo Metropolitan Institute of Gerontology, Tokyo, Japan

Takayuki SumidaDepartment of Internal Medicine, Faculty of Medicine, University of Tsukuba, Tsukuba, Japan;

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Anti-cyclic citrullinated glucose-6-phosphate isomerase peptide-7 (CCG-7) antibodies were suppressed by biologics treatment and deposited to citrullinated proteins in CD68-positive cells in the RA synovium

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Anti-cyclic citrullinated glucose-6-phosphate isomerase peptide-7 (CCG-7) antibodies were suppressed by biologics treatment and deposited to citrullinated proteins in CD68-positive cells in the RA synovium

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