Abstract
The authors report an unusual case of exophthalmos, deficiency of the VIth nerve and of bilateral optic atrophy in a patient affected by intracranial meningiomatosis. The problem relating to the mechanism of development of exophthalmos is dealt with. Those cases in which direct invasion of the orbital cavity can be excluded with certainty indicate pathological compression on the cavernous sinus as the mechanism responsible for the symptomatology.