Calprotectin (S100A8/A9), S100A12, and EDTA-resistant S100A12 complexes (ERAC) in primary Sjögren’s syndrome

HH NordalDepartment of Clinical Science, University of Bergen, Oslo, Norway;Department of Rheumatology, Haukeland University Hospital, Bergen, NorwayCorrespondence[email protected]

JG BrunDepartment of Clinical Science, University of Bergen, Oslo, Norway;Department of Rheumatology, Haukeland University Hospital, Bergen, Norway

A-K HalseDepartment of Clinical Science, University of Bergen, Oslo, Norway;Department of Rheumatology, Haukeland University Hospital, Bergen, Norway

TM MadlandDepartment of Rheumatology, Haukeland University Hospital, Bergen, Norway

MK FagerholProf. Fagerhol’s Research Laboratory, Oslo, Norway

R JonssonDepartment of Clinical Science, University of Bergen, Oslo, Norway;Department of Rheumatology, Haukeland University Hospital, Bergen, Norway

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Acknowledgements

We thank M Eidsheim for technical assistance with the analyses of ERAC and A Larsen for the analyses of calprotectin and S100A12. This study was supported by GC Rieber Funds, the Broegelmann Foundation, the Marie and Else Mustad Legacy and the Norwegian Rheumatism Association.

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Calprotectin (S100A8/A9), S100A12, and EDTA-resistant S100A12 complexes (ERAC) in primary Sjögren’s syndrome

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Calprotectin (S100A8/A9), S100A12, and EDTA-resistant S100A12 complexes (ERAC) in primary Sjögren’s syndrome

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