A compound T60A and V122I heterozygosity in the transthyretin gene causing early onset severe cardiac amyloidosis

Yi-Chang LiuMyeloma Institute for Research and Therapy, University of Arkansas for Medical Sciences Little Rock, ARUSA;Faculty of Medicine, College of Medicine, Kaohsiung Medical University KaohsiungTaiwan;Department of Hematology-Oncology, Kaohsiung Medical University Hospital KaohsiungTaiwan

Honey Vodur ReddiMolecular Genetics Laboratory, Department of Laboratory Medicine and Pathology, Mayo Clinic Rochester, MNUSA

Sarah WaheedMyeloma Institute for Research and Therapy, University of Arkansas for Medical Sciences Little Rock, ARUSA

Daisy AlapatDepartment of Pathology, University of Arkansas for Medical Sciences Little Rock, ARUSA

W. Edward HighsmithMolecular Genetics Laboratory, Department of Laboratory Medicine and Pathology, Mayo Clinic Rochester, MNUSA

Ricky D. EdmondsonMyeloma Institute for Research and Therapy, University of Arkansas for Medical Sciences Little Rock, ARUSA

Bart BarlogieMyeloma Institute for Research and Therapy, University of Arkansas for Medical Sciences Little Rock, ARUSA

Frits van RheeMyeloma Institute for Research and Therapy, University of Arkansas for Medical Sciences Little Rock, ARUSACorrespondence[email protected]

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"A compound T60A and V122I heterozygosity in the transthyretin gene causing early onset severe cardiac amyloidosis." Amyloid, 21(2), pp. 134–135

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A compound T60A and V122I heterozygosity in the transthyretin gene causing early onset severe cardiac amyloidosis

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A compound T60A and V122I heterozygosity in the transthyretin gene causing early onset severe cardiac amyloidosis

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