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Original

Genomics and the future of pharmacotherapy in psychiatry

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Pages 523-530 | Published online: 11 Jul 2009

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Jian-Ping Zhang & Anil K Malhotra. (2013) Pharmacogenetics of antipsychotics: recent progress and methodological issues. Expert Opinion on Drug Metabolism & Toxicology 9:2, pages 183-191.
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John M. Kane & Christoph U. Correll. (2010) Pharmacologic treatment of schizophrenia. Dialogues in Clinical Neuroscience 12:3, pages 345-357.
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Ramón Cacabelos. (2009) Pharmacogenomics and therapeutic strategies for dementia. Expert Review of Molecular Diagnostics 9:6, pages 567-611.
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Jessica L. Bourdon, Rachel A. Davies & Elizabeth C. Long. (2020) Four Actionable Bottlenecks and Potential Solutions to Translating Psychiatric Genetics Research: An Expert Review. Public Health Genomics 23:5-6, pages 171-183.
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S M Bahr, B C Tyler, N Wooldridge, B D Butcher, T L Burns, L M Teesch, C L Oltman, M A Azcarate-Peril, J R Kirby & C A Calarge. (2015) Use of the second-generation antipsychotic, risperidone, and secondary weight gain are associated with an altered gut microbiota in children. Translational Psychiatry 5:10, pages e652-e652.
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J Kambeitz, M Romanos & U Ettinger. (2013) Meta-analysis of the association between dopamine transporter genotype and response to methylphenidate treatment in ADHD. The Pharmacogenomics Journal 14:1, pages 77-84.
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Britt I. Drögemöller, Galen E.B. Wright, Dana J.H. Niehaus, Robin Emsley & Louise Warnich. (2013) Next-generation sequencing of pharmacogenes. Pharmacogenetics and Genomics 23:12, pages 666-674.
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R. De Vecchis, C. Esposito & C. Ariano. (2013) Cabergoline use and risk of fibrosis and insufficiency of cardiac valvesCabergolingabe und Risiko einer Fibrose mit Herzklappeninsuffizienz. Herz 38:8, pages 868-880.
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Firoza Mamdani, Maureen V. Martin, Todd Lencz, Brandi Rollins, Delbert G. Robinson, Emily A. Moon, Anil K. Malhotra & Marquis P. Vawter. (2013) Coding and Noncoding Gene Expression Biomarkers in Mood Disorders and Schizophrenia. Disease Markers 35, pages 11-21.
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Ramón Cacabelos. (2012) Pharmacogenomics of Central Nervous System (CNS) Drugs. Drug Development Research 73:8, pages 461-476.
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K. G. Claw, R. Y. Tito, A. C. Stone & B. C. Verrelli. (2010) Haplotype Structure and Divergence at Human and Chimpanzee Serotonin Transporter and Receptor Genes: Implications for Behavioral Disorder Association Analyses. Molecular Biology and Evolution 27:7, pages 1518-1529.
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S Le Hellard, T W Mühleisen, S Djurovic, J Fernø, Z Ouriaghi, M Mattheisen, C Vasilescu, M B Raeder, T Hansen, J Strohmaier, A Georgi, F F Brockschmidt, I Melle, I Nenadic, H Sauer, M Rietschel, M M Nöthen, T Werge, O A Andreassen, S Cichon & V M Steen. (2008) Polymorphisms in SREBF1 and SREBF2, two antipsychotic-activated transcription factors controlling cellular lipogenesis, are associated with schizophrenia in German and Scandinavian samples. Molecular Psychiatry 15:5, pages 463-472.
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Sofia I. I. Kring, Thomas Werge, Claus Holst, Søren Toubro, Arne Astrup, Torben Hansen, Oluf Pedersen & Thorkild I. A. Sørensen. (2009) Polymorphisms of Serotonin Receptor 2A and 2C Genes and COMT in Relation to Obesity and Type 2 Diabetes. PLoS ONE 4:8, pages e6696.
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Eduard Maron, Anu Tammiste, Kristi Kallassalu, Triin Eller, Veiko Vasar, David J. Nutt & Andres Metspalu. (2009) Serotonin transporter promoter region polymorphisms do not influence treatment response to escitalopram in patients with major depression. European Neuropsychopharmacology 19:6, pages 451-456.
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Charles E Dean. (2009) Personalized Medicine: Boon or Budget-Buster?. Annals of Pharmacotherapy 43:5, pages 958-962.
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Ramón Cacabelos. 2009. The Handbook of Neuropsychiatric Biomarkers, Endophenotypes and Genes. The Handbook of Neuropsychiatric Biomarkers, Endophenotypes and Genes 3 63 .
Kenneth Blum, Amanda LihChuan Chen, Thomas JH Chen, Eric R Braverman, Jeffrey Reinking, Seth H Blum, Kimberly Cassel, Bernard W Downs, Roger L Waite, Lonna Williams, Thomas J Prihoda, Mallory M Kerner, Tomas Palomo, David E Comings, Howard Tung, Patrick Rhoades & Marlene Oscar-Berman. (2008) Activation instead of blocking mesolimbic dopaminergic reward circuitry is a preferred modality in the long term treatment of reward deficiency syndrome (RDS): a commentary. Theoretical Biology and Medical Modelling 5:1.
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