The latency-associated transcript of herpes simplex virus type 1 promotes survival and stimulates axonal regeneration in sympathetic and trigeminal neurons

Mohamed A Hamza Departments of Pharmacology and Toxicology

Dennis M Higgins Departments of Pharmacology and Toxicology

Lawrence T Feldman Department of Microbiology, Immunology and Molecular Genetics, David Geffen School of Medicine at UCLA, Los Angeles, California, USA

William T Ruyechan Microbiology and Immunology, University at Buffalo, State University of New York, Buffalo, New York, USA

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Isamu Mori. (2010) Herpes simplex virus type 1 persists in the aged brain through hypothetical expression of accessory genes. Journal of Neurovirology 16:3, pages 203-207.
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Hassanain S. Toma, Andrea T. Murina, Raymond G. Areaux$suffix/text()$suffix/text(), Donna M. Neumann, Partha S. Bhattacharjee, Timothy P. Foster, Herbert E. Kaufman & James M. Hill. (2008) Ocular HSV-1 Latency, Reactivation and Recurrent Disease. Seminars in Ophthalmology 23:4, pages 249-273.
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The latency-associated transcript of herpes simplex virus type 1 promotes survival and stimulates axonal regeneration in sympathetic and trigeminal neurons

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The latency-associated transcript of herpes simplex virus type 1 promotes survival and stimulates axonal regeneration in sympathetic and trigeminal neurons

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