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Scandinavian Journal of Rheumatology Volume 38, 2009 - Issue 6
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Articles

S100A9 is not essential for disease expression in an acute (K/BxN) or chronic (CIA) model of inflammatory arthritis

RR Rampersad Thurston Arthritis Research Center, University of North Carolina School of Medicine, Division of Rheumatology, Allergy, and Immunology, Chapel Hill, NC
,
D Esserman University of North Carolina School of Medicine, Division of General Medicine and Clinical Epidemiology, Chapel Hill, NC
,
MW McGinnis Thurston Arthritis Research Center, University of North Carolina School of Medicine, Division of Rheumatology, Allergy, and Immunology, Chapel Hill, NC
,
DM Lee Division of Rheumatology, Allergy, and Immunology, Brigham and Women's Hospital, Boston, MA, USA
,
DD Patel Thurston Arthritis Research Center, University of North Carolina School of Medicine, Division of Rheumatology, Allergy, and Immunology, Chapel Hill, NC; Novartis Institutes for BioMedical Research, Basel, Switzerland
&
TK Tarrant Thurston Arthritis Research Center, University of North Carolina School of Medicine, Division of Rheumatology, Allergy, and Immunology, Chapel Hill, NCCorrespondence[email protected]
Pages 445-449 | Published online: 18 Nov 2009

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MC Melnyk, I Shalev, J Zhang, A Bartczak, RM Gorczynski, N Selzner, R Inman, PA Marsden, MJ Phillips, DA Clark & GA Levy. (2011) The prothrombinase activity of FGL2 contributes to the pathogenesis of experimental arthritis. Scandinavian Journal of Rheumatology 40:4, pages 269-278.
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Articles from other publishers (7)

Irene Di Ceglie, Peter L. E. M. van Lent, Edwin J. W. Geven, Marije I. Koenders, Arjen B. Blom, Thomas Vogl, Johannes Roth & Martijn H. J. van den Bosch. (2021) S100A8/A9 is not essential for the development of inflammation and joint pathology in interleukin-1 receptor antagonist knockout mice. Arthritis Research & Therapy 23:1.
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N. Jung, J.-L. Bueb, F. Tolle & S. Bréchard. (2019) Regulation of neutrophil pro-inflammatory functions sheds new light on the pathogenesis of rheumatoid arthritis. Biochemical Pharmacology 165, pages 170-180.
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Derek T. Holyoak, Ye F. Tian, Marjolein C. H. van der Meulen & Ankur Singh. (2016) Osteoarthritis: Pathology, Mouse Models, and Nanoparticle Injectable Systems for Targeted Treatment. Annals of Biomedical Engineering 44:6, pages 2062-2075.
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Reza Akbarzadeh, Xinhua Yu, Thomas Vogl, Ralf J. Ludwig, Enno Schmidt, Detlef Zillikens & Frank Petersen. (2016) Myeloid-related proteins-8 and -14 are expressed but dispensable in the pathogenesis of experimental epidermolysis bullosa acquisita and bullous pemphigoid. Journal of Dermatological Science 81:3, pages 165-172.
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Teresa K. Tarrant, Peng Liu, Rishi R. Rampersad, Denise Esserman, Lisa R. Rothlein, Roman G. Timoshchenko, Marcus W. McGinnis, David J. Fitzhugh, Dhavalkumar D. Patel & Alan M. Fong. (2012) Decreased Th17 and antigen‐specific humoral responses in CX 3 CR1‐deficient mice in the collagen‐induced arthritis model . Arthritis & Rheumatism 64:5, pages 1379-1387.
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Michelle M. AverillShelley BarnhartLev BeckerXin LiJay W. HeineckeRenee C. LeBoeufJessica A. HamermanClemens SorgClaus KerkhoffKarin E. Bornfeldt. (2011) S100A9 Differentially Modifies Phenotypic States of Neutrophils, Macrophages, and Dendritic Cells. Circulation 123:11, pages 1216-1226.
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Peter L. van Lent, Lilyanne C. Grevers, Rik Schelbergen, Arjen Blom, Jeroen Geurts, Annet Sloetjes, Thomas Vogl, Johannes Roth & Wim B. van den Berg. (2010) S100A8 causes a shift toward expression of activatory Fcγ receptors on macrophages via toll-like receptor 4 and regulates Fcγ receptor expression in synovium during chronic experimental arthritis. Arthritis & Rheumatism 62:11, pages 3353-3364.
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