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Marie-Eve Piché & Paul Poirier. (2018) Obesity, ectopic fat and cardiac metabolism. Expert Review of Endocrinology & Metabolism 13:4, pages 213-221.
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Kannayiram Alagiakrishnan, Maciej Banach, Ali Ahmed & Wilbert S. Aronow. (2016) Complex relationship of obesity and obesity paradox in heart failure – higher risk of developing heart failure and better outcomes in established heart failure. Annals of Medicine 48:8, pages 603-613.
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Julie. Lessard & André. Tchernof. (2012) Depot- and obesity-related differences in adipogenesisAdipocyte hypertrophy and hyperplasia are known to facilitate lipid storage in adipose tissues by increasing adipocyte cell size and number, respectively. Adipogenesis is the process resulting in adipose tissue hyperplasia. Although depot-specific differences and obesity-related modulation of adipocyte size are well documented, available data on adipogenesis and adipose tissue hyperplasia are less conclusive. Most studies support a reduction of adipogenesis in the obese state. Preadipocytes of the subcutaneous fat depot appear to be more responsive to adipogenic stimulation compared with those from visceral fat compartments in most studies. A number of studies support the notion that adipose tissue expansion through hyperplasia reduces ectopic lipid excess and obesity-related complications. Several genetic variants have been identified in the genes coding for adipogenesis-regulating proteins. While some of these variants have been clearly associated with the phenotypes of obesity and obesity-related alterations, available data highlight the importance of considering gene–gene and gene–diet interactions.. Clinical Lipidology 7:5, pages 587-596.
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