Figures & data
Figure 1. Effects of administration of different doses of the dry residue from ethanol extract of P. carpunya on gastric damage induced by diclofenac treatment. +, P < 0.5; + +, P < 0.01 versus diclofenac group.
Figure 2. Microscopic study (H/E section) of gastric mucosa of animals pretreated with dry residue from ethanol extract of P. carpunya before ulcer induction with diclofenac. (A and B), gastric lesion after diclofenac administration; (C and D), gastric mucosa of rats treated with P. carpunya (250 mg/kg) before diclofenac administration. Original magnification × 100 and × 200 respectively.
Figure 3. Effects of administration of different doses of the dry residue from ethanol extract of P. carpunya on myeloperoxidase activity in gastric damage induced by diclofenac treatment. + +, P < 0.01 versus diclofenac group.
Figure 4. Effects of administration of different doses of the dry residue from ethanol extract of P. carpunya on myeloperoxidase activity on rat peritoneal leukocytes treated with calcium ionophore A23187. Caffeic acid was included as a reference compound. **, P < 0.01 versus A23187 group.
Figure 5. Effects of administration of different doses of the dry residue from ethanol extract of P. carpunya on lipid peroxidation, superoxide dismutase and glutathione peroxidase activity in gastric damage induced by diclofenac treatment. *, P < 0.05; and **, P < 0.01 versus sham group; +, P < 0.05 versus diclofenac group.
Figure 6. Effects of administration of different doses of the dry residue from ethanol extract of P. carpunya on prostaglandin E2 levels in gastric damage induced by diclofenac treatment. *, P < 0.05 versus sham group.
