The Effect of Exercise Training on the Responsiveness of Renal Resistance Arteries in Rats

Figures & data

Figure 1. Cumulative concentration–response curves to acetylcholine (A), adenosine (B), bradykinin (C), isoproterenol (D), and sodium nitroprusside (E) in renal resistance arteries from experimental groups.

Note: Values are means ± SE.

Figure 2. Cumulative concentration–response curves to norepinephrine (A), potassium chloride (B), endothelin (C), thromboxane A₂ (D), and vasopressin (E) in renal resistance arteries from experimental groups.

Notes: Values are means ± SE.

*Denotes p < 0.01, difference from control group.

Table 1. Maximal dilation response (Emax) values to vasodilator agents in isolated renal resistance arteries in experimental rats.

Agents (%)	Control	Exercise
Acetylcholine	76.5 ± 4.48	75.5 ± 4.33
Adenosine	68.2 ± 5.84	71.6 ± 5.43
Bradykinin	65.6 ± 8.0	60.2 ± 6.23
Sodium nitroprusside	95.7 ± 1.37	97.2 ± 0.94
Isoproterenol	70.1 ± 5.5	69.4 ± 6.8

Note: Values are means ± SE.

Table 2. Maximal constriction response (Emax) values to vasodilator agents in isolated renal resistance arteries in experimental rats.

Agents (g)	Control	Exercise
Norepinephrine	1.82 ± 0.28	2.56 ± 0.19
Thromboxane A2	1.58 ± 0.19	1.84 ± 0.15
Potassium chloride	2.13 ± 0.29	2.33 ± 0.17
Endothelin	1.71 ± 0.07	1.89 ± 0.06
Vasopressin	1.97 ± 0.23	1.96 ± 0.29

Notes: Values are means ± SE.

*Denotes p < 0.05, difference from control group.