Poststreptococcal glomerulonephritis in children with congenital anomalies of the kidney and urinary tract

Figures & data

Figure 1. Tc⁹⁹mDMSA scan shows solitary left kidney.

Table 1. Clinical and laboratory features of five patients with CAKUT who suffered from acute poststreptococcal glomerulonephritis.

Patient	Sex	Age at diagnosis of CAKUT/ PSGN (years)	CAKUT phenotype	Cr before PSGN (μmol/L)	Proteinuria at PSGN onset (g/24 h)	Cr during PSGN (μmol/L)	C3 during PSGN (g/L)	C3 after 12 weeks (g/L)	Follow up (years)	BP at the last follow-up (mmHg)	Proteinuria at the last follow up (g/24 h)	Cr at the last follow-up (μmol/L)
1.	M	16/16	Right renal agenesis	NK	0.44	154	0.23	1.43	6	122/66	1.63	88
2.	F	11/11	Bill hypodysplasia	NK	0.77	253	0.33	0.99	7	130/90	0.98	423
3.	F	2/4	Bill VUR, Right RN	36	1+	46	0.45	1.03	9	96/68	0.11	40
4.	M	15/15	Left kidney ectopic,  hypo/dysplastic	NK	0.33	69	0.50	1.22	5	108/80	0.06	74
5.	M	7/12	Left UPJ obstruction	43	0.43	78	0.24	1.33	5	122/62	0.04	45

Notes: M, male; F, female; NK, not known; Cr, creatinine; CAKUT, congenital anomaly of the kidney and urinary tract; PSGN, poststretococcal glomerulonephritis; VUR, vesicoureteral reflux; RN, reflux nephropathy; UPJ, ureteropelvic junction; BP, blood pressure.

Figure 2. Ultrasound scan reveals small right kidney with hyperechogenic parenchyma and loss of corticomedullary differentiation.

Figure 3. Tc⁹⁹mDMSA scan shows hypodisplastic ectopic left kidney.