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Leukemia & Lymphoma Volume 59, 2018 - Issue 5
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Letters to the Editor

Mutational frequencies of CD79B and MYD88 vary greatly between primary testicular DLBCL and gastrointestinal DLBCL

Mareike FrickDepartment of Hematology, Oncology and Tumor Immunology, and Berlin Institute of Health, Charité – Universitätsmedizin Berlin, Berlin, Germany; ;Berlin Institute of Health (BIH), BIH Biomedical Innovation Academy, Berlin, Germany; Correspondence[email protected]
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Marcus BettstetterMolekularpathologie Südbayern, Munich, Germany; View further author information
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Simone BertzInstitute of Pathology, University Hospital Erlangen, Erlangen, Germany; View further author information
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Stephan Schwarz-FurlanInstitute of Pathology, University Hospital Erlangen, Erlangen, Germany; ;Institute of Pathology, Klinikum Kaufbeuren, Kaufbeuren, Germany; View further author information
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Arndt HartmannInstitute of Pathology, University Hospital Erlangen, Erlangen, Germany; View further author information
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Thomas RichterInstitute of Pathology, Rosenheim, Germany; View further author information
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Dido LenzeDepartment of Pathology, Charité-Universitätsmedizin, Berlin, Germany; View further author information
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Michael HummelDepartment of Pathology, Charité-Universitätsmedizin, Berlin, Germany; View further author information
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Martin DreylingDepartment of Internal Medicine III, University Hospital Munich, Munich, Germany; View further author information
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Georg LenzTranslational Oncology, Department of Medicine A, University Hospital Münster, Münster, Germany; ;Cluster of Excellence EXC 1003, Cells in Motion, Münster, GermanyView further author information
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Andreas GaumannInstitute of Pathology, Klinikum Kaufbeuren, Kaufbeuren, Germany; View further author information
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Pages 1260-1263 | Received 23 Dec 2016, Accepted 13 Aug 2017, Published online: 03 Sep 2017
 
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Potential conflict of interest

Disclosure forms provided by the authors are available with the full text of this article online at https://doi.org:10.1080/10428194.2017.1370546.

Additional information

Funding

This work was supported by individual research grants to G.L. from the Deutsche Krebshilfe, the Else Kroener-Fresenius-Stiftung, the Swiss National Science Foundation (Sinergia grant), the Deutsche Forschungsgemeinschaft, and Novartis pharma. In addition, this work was also supported by the Deutsche Forschungsgemeinschaft, DFG EXC 1003 Cells in Motion – Cluster of Excellence, Muenster, Germany. Further, this work was supported by institutional funding money to M.H. M.F. is a participant of the Charité Clinician Scientist Program funded by the Charité University Medical Center Berlin and the Berlin Institute of Health.

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