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The World Journal of Biological Psychiatry Volume 9, 2008 - Issue 3
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Brief Reports

Complement C4B protein in schizophrenia

Karine R. Mayilyan MRC Immunochemistry Unit, Department of Biochemistry, University of Oxford, Oxford, UK; Institute of Molecular Biology, Armenian National Academy of Sciences, Yerevan, ArmeniaCorrespondence[email protected]
,
Alister W. Dodds MRC Immunochemistry Unit, Department of Biochemistry, University of Oxford, Oxford, UK
,
Anna S. Boyajyan Institute of Molecular Biology, Armenian National Academy of Sciences, Yerevan, Armenia
,
Armen F. Soghoyan Department of Psychiatry and Medical Psychology, Yerevan State Medical University, Armenian Health Ministry, Yerevan, Armenia
&
Robert B. Sim MRC Immunochemistry Unit, Department of Biochemistry, University of Oxford, Oxford, UKCorrespondence[email protected]
Pages 225-230 | Received 20 Jun 2006, Published online: 12 Jul 2009
 
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Abstract

Partial and/or complete deficiency of the complement protein C4 is associated with autoimmune and infectious diseases. Infectious or autoimmune processes may have a role in schizophrenia. Previous reports suggest abnormalities in the complement C4B isotype in schizophrenia and other mental disorders. We assessed C4A and C4B isotypes and serum C4B protein concentration in Armenian schizophrenic patients. Although there was no difference in frequency of C4BQ0, C4B serum protein level was significantly decreased in the schizophrenic patients compared with healthy controls.

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