Presence of Gastric Autoantibodies Impairs Gastric Secretory Function in Patients with Helicobacter pylori-positive Duodenal Ulcer

References

• Negrini R, Lisato L, Zanella I, et al. H. pylori infection induces antibodies cross-reactive with human gastric mucosa. Gastroenterology 1991;101: 437–45.
   Google Scholar
• Faller G, Steininger H, Kranzlein J, et al. Antigastric autoantibodies in Helicobacter pylori infection: implications of histological and clinical parameters of gastritis. Gut 1997;41: 619–23.
   PubMed Web of Science ®Google Scholar
• Negrini R, Savio A, Poiesi C, et al. Antigenic mimicry between Helicobacter pylori and gastric mucosa in the pathogenesis of body atrophic gastritis. Gastroenterology 1996;111:655–65.
   PubMed Web of Science ®Google Scholar
• Faller G, Winter M, Steininger H, et al. Decrease of antigastric autoantibodies after cure of H. pylori infection. Gut 1997; 41 Suppl 1:A61–2.
   Google Scholar
• Steininger H, Faller G, Dewald Brabletz T, et al. A rate of apoptosis in chronic gastritis and its correlation with antigastric autoantibodies. Virchows Arch 1998;433:13–8.
   PubMed Web of Science ®Google Scholar
• Berstad AE, Hatlebakk JG, Maartmann-Moe H, et al. Helicobacter pylori gastritis and epithelial cell proliferation in patients with reflux oesophagitis after treatment with lansoprazole. Gut 1997;41:740–7.
   Google Scholar
• Faller G, Winter M, Steininger H, et al. Antigastric autoantibodies and gastric secretory function in Helicobacter pylori-infected patients with duodenal ulcer and non-ulcer dyspepsia. Scand J Gastroenterol 1998;33:276–82.
   PubMed Web of Science ®Google Scholar
• Price AB The Sydney system: histologic division. Eur J Gastroenterol Hepatol 1991,6:209–22.
   Google Scholar
• Parente F, Imbesi V, Maconi G, et al. Influence of bacterial CagA status on gastritis, gastric function indices, and pattern of symptoms in H. pylori-positive dyspeptic patients. Am J Gastroenterol 1998;93:1073–9.
   PubMedGoogle Scholar
• Parente F, Maconi G, Sangaletti O, et al. Prevalence of Helicobacter pylori infection and related gastroduodenal lesions in spouses of Helicobacter pylori positive duodenal ulcer patients. Gut 1996;39:629–33.
   PubMed Web of Science ®Google Scholar
• Chittajallu RS, Ardill JES, McColl KEL Eradicating Helicobacter pylori infection lowers gastrin mediated acid secretion by two thirds in patients with duodenal ulcer. Gut 1993;34:1060–5.
   PubMedGoogle Scholar
• El-Omar E, Penman ID, Ardill JES, et al. Helicobacter pylori and abnormalities of acid secretion in patients with duodenal ulcer disease. Gastroenterology 1995;109: 681–91.
   Google Scholar
• Parente F, Maconi G, Sangaletti O, et al. Behaviour of acid secretion, gastrin release, serum pepsinogen I, and gastric emptying of liquids over six months from eradication of Helicobacter pylori in duodenal ulcer patients. A controlled study. Gut 1995,37:210–15.
   PubMed Web of Science ®Google Scholar
• El-Omar E, Penman I, Ardill JE, et al. A substantial proportion of non-ulcer dyspepsia patients have the same abnormality of acid secretion as duodenal ulcer patients. Gut 1995,36:534–8.
   Google Scholar
• Harris AW, Gummett PA, Misiewicz JJ, et al. Eradication of Helicobacter pylori in patients with duodenal ulcer lowers basal and peak acid output to gastrin releasing peptide and pentagastrin. Gut 1996;38:663–7.
   PubMed Web of Science ®Google Scholar
• El-Omar E, Oien K, El-Nujumi A, et al. Helicobacter pylori infection and chronic gastric acid hyposecretion. Gastroenterology 1997;113:15–24.
   PubMed Web of Science ®Google Scholar
• McColl KEL, El-Omar E, Gillen D. Alterations in gastric physiology in Helicobacter pylori infection: causes of different diseases or all epiphenomena? Ital J Gastroenterol Hepatol 1997;29: 459–64.
   Google Scholar
• Figura N. Helicobacter pylori exotoxins and gastroduodenal diseases associated with cytotoxic strain infection. Aliment Pharmacol Ther 1996;10 Suppl 1:79–96.
   PubMed Web of Science ®Google Scholar
• Appelmelk BJ, Simoons-Smit I, Negrini R, et al. Potential role of molecular mimicry between Helicobacter pylori lipopolysaccharide and host Lewis blood group antigens in autoimmunity. Infect Immun 1996;64:2031–40.
   PubMed Web of Science ®Google Scholar
• Lam SK. Pathogenesis and pathophysiology of duodenal ulcer. Clin Gastroenterol 1984;13:447–72.
   PubMedGoogle Scholar
• Burman P, Mardh S, Norberg L Parietal cell autoantibodies in pernicious anemia inhibit H,K-adenosine triphosphatase, the proton pump of the stomach. Gastroenterology 1989;96:1484–8.
   Google Scholar
• Wallace JL, Cucala M, Mugridge K, et al Secretagogue-specifi c effects of interleukin-1 on gastric acid secretion. Am J Physiol 1991;261:G559–64.
   PubMed Web of Science ®Google Scholar
• Covacci A. Biological marker for diseases sustained by Helicobacter pylori infection. Digest Liver Dis 2000; 32:384–5.
   Google Scholar