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Scandinavian Journal of Gastroenterology Volume 40, 2005 - Issue 5
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Correspondence

Decreased mitochondrial oxidative capacity in hereditary tyrosinemia type 1

Donato Rigante Department of Pediatric Sciences, Università Cattolica Sacro Cuore, Rome, ItalyCorrespondence[email protected]
,
Antonio Gasbarrini Department of Internal Medicine, Università Cattolica Sacro Cuore, Rome, Italy
,
Enrico Celestino Nista Department of Internal Medicine, Università Cattolica Sacro Cuore, Rome, Italy
&
Marcello Candelli Department of Internal Medicine, Università Cattolica Sacro Cuore, Rome, Italy
Pages 612-613 | Published online: 08 Jul 2009

References

  • Kvittingen EA. Tyrosinaemia: treatment and outcome. J Inherited Metab Dis 1995;18:375–9.
  • Becker M. 13C breath tests for measurement of liver function. Gut 1998;43 Suppl 3:25–7.
  • Lauterburg BH, Liang D, Schwarzenbach FA, Breen KJ. Mitochondrial dysfunction in alcoholic patients as assessed by breath analysis. Hepatology 1993;17:418–22.
  • Elsas LJ, Ellerine NP, Klein PD. Practical methods to estimate whole body leucine oxidation in maple syrup urine disease. Pediatr Res 1993;33:445–51.

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