REFERENCES
- Candenas, L., Seda, M., Noheda, P., Buschmann, H., Cintado, C.G., Martin, J.D., &. Pinto, F.M. (2006). Molecular diversity of voltage-gated sodium channel alpha and beta subunit mRNAs in human tissues. Eur. J. Pharmacol. 541, 9–16.
- Catterall, W.A. (2000). From ionic currents to molecular mechanisms: The structure and function of voltage-gated sodium channels. Neuron 26, 13–25.
- Celesia, G.G. (2001). Disorders of membrane channels or channelopathies. Clin. Neurophysiol. 112, 2–18.
- Chen, Y.H., Dale, T.J., Romanos, M.A., Whitaker, W.R., Xie, X.M., &. Clare, J.J. (2000). Cloning, distribution and functional analysis of the type III sodium channel from human brain. Eur. J. Neurosci. 12, 4281–4289.
- Donahue, L.M., Coates, P.W., Lee, V.H., Ippensen, D.C., Arze, S.E., &. Poduslo, S.E. (2000). The cardiac sodium channel mRNA is expressed in the developing and adult rat and human brain. Brain. Res. 887, 335–343.
- George, A.L. (2005). Inherited disorders of voltage-gated sodium channels. J. Clin. Invest. 115, 1990–1999.
- Goldin, A.L. (2001). Resurgence of sodium channel research. Annu. Rev. Physiol. 63, 871–894.
- Hartmann, H.A., Colom, L.V., Sutherland, M.L., &. Noebels, J.L. (1999). Selective localization of cardiac SCN5A sodium channels in limbic regions of rat brain. Nat. Neurosci. 2, 593–595.
- Jacobs, M.P., Fischbach, G.D., Davis, M.R., Dichter, M.A., Dingledine, R., Lowenstein, D.H., Morrell, M.J., Noebels, J.L., Rogawski, M.A., Spencer, S.S., &. Theodore, W.H. (2001). Future directions for epilepsy research. Neurology 57, 1536–1542.
- Kerr, N.C., Holmes, F.E., &. Wynick, D. (2004). Novel isoforms of the sodium channels Nav1.8 and Nav1.5 are produced by a conserved mechanism in mouse and rat. J. Biol. Chem. 279, 24826–24833.
- Kohling, R. (2002). Voltage-gated sodium channels in epilepsy. Epilepsia 43, 1278–1295.
- Mechaly, I., Scamps, F., Chabbert, C., Sans, A., &. Valmier, J. (2005). Molecular diversity of voltage-gated sodium channel alpha subunits expressed in neuronal and non-neuronal excitable cells. Neuroscience 130, 389–396.
- Meisler, M.H. &. Kearney, J.A. (2005). Sodium channel mutations in epilepsy and other neurological disorders. J. Clin. Invest. 115, 2010–2017.
- Meisler, M.H., Kearney, J., Ottman, R., &. Escayg, A. (2001). Identification of epilepsy genes in human and mouse. Annu. Rev. Genet. 35, 567–588.
- Muzny, D.M., Scherer, S.E., Kaul, R., Wang, J., Yu, J., , et al. (2006). The DNA sequence, annotation and analysis of human chromosome 3. Nature 440, 1194–1198.
- Ou, S.W., Kameyama, A., Hao, L.Y., Horiuchi, M., Minobe, E., Wang, W.Y., Makita, N., &. Kameyama, M. (2005). Tetrodotoxin-resistant Na+ channels in human neuroblastoma cells are encoded by new variants of Nav1.5/SCN5A. Eur. J. Neurosci. 22, 793–801.
- Raymond, C.K., Castle, J., Garrett-Engele, P., Armour, C.D., Kan, Z., Tsinoremas, N., &. Johnson, J.M. (2004). Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia. J. Biol. Chem. 279, 46234–46241.
- Tan, J., Liu, Z., Nomura, Y., Goldin, A.L., &. Dong, K. (2002). Alternative splicing of an insect sodium channel gene generates pharmacologically distinct sodium channels. J. Neurosci. 22, 5300–5309.
- Ure, J.A. &. Perassolo, M. (2000). Update on the pathophysiology of epilepsies. J. Neurol. Sci. 177, 1–17.
- Wang, Q., Li, Z.Z., &. Shen, J.X. (1996). Genomic organization of the human SCN5A gene encoding the cardiac sodium channel. Genomics 34, 9–16.
- Ye, B., Valdivia, C.R., Ackerman, M.J., &. Makielski, J.C. (2003). A common human SCN5A polymorphism modifies expression of an arrhythmia causing mutation. Physiol. Genomics 12, 187–193.
- Yu, F.H. &. Catterall, W.A. (2003). Overview of the voltage-gated sodium channel family. Genome Biol. 4, 207.1–207.7.
- Zimmer, T., Bollensdorff, C., Haufe, V., Birch-Hirschfeld, E., &. Benndorf, K. (2002). Mouse heart Na+ channels: primary structure and function of two isoforms and alternatively spliced variants. Am. J. Physiol. Heart Circ. Physiol. 282, H1007–H1017.