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Scandinavian Journal of Rheumatology Volume 35, 2006 - Issue 2
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LetterToEditor

The genetic contribution of CTLA‐4 dimorphisms in promoter and exon 1 regions in Japanese patients with rheumatoid arthritis

F. Takeuchi Department of Internal Medicine (Allergy and Rheumatology), Faculty of Medicine, University of Tokyo, TokyoCorrespondence[email protected]
,
K. Kawasugi Department of Internal Medicine (Allergy and Rheumatology), Faculty of Medicine, University of Tokyo, Tokyo
,
M. Mori Department of Internal Medicine (Allergy and Rheumatology), Faculty of Medicine, University of Tokyo, Tokyo
,
N. Nakaue Department of Internal Medicine (Allergy and Rheumatology), Faculty of Medicine, University of Tokyo, Tokyo
,
N. Kobayashi Department of Internal Medicine (Allergy and Rheumatology), Faculty of Medicine, University of Tokyo, Tokyo
,
S. Kuwata 3rd Department of Internal Medicine, Faculty of Medicine, Teikyo University School of Medicine, Ichihara
,
T. Murayama Johoku Clinic, Kanazawa
&
K. Matsuta Matsuta Clinic, Tokyo, Japan
 show all
Pages 154-155 | Received 14 Apr 2005, Accepted 02 Sep 2005, Published online: 25 Aug 2009

References

  • Seidl C., Donner H., Fischer B., Usadel K. H., Seifried E., Keltwasser J. P., et al. CTLA4 codon 17 dimorphism in patients with rheumatoid arthritis. Tissue Antigens 1998; 51: 62–6
  • Yanagawa T., Gomi K., Nakao E., Inada S. CTLA4 gene polymorphism in Japanese patients with rheumatoid arthritis. J Rheumatol 2000; 27: 2740–2
  • Lee C. S., Lee Y. J., Liu H. F., Su C. H., Chang S. C., Wang B. R., et al. Association of CTLA4 gene A–G polymorphism with rheumatoid arthritis in Chinese. Clin Rheumatol 2003; 22: 221–4
  • Barton A., Myerscough A., John S., Gonzalez‐Gay M., Ollier W., Worthington J. A single nucleotide polymorphism in exon 1 of cytotoxic T‐lymphocyte‐associated‐4 (CTLA‐4) is not associated with rheumatoid arthritis. Rheumatology 2000; 39: 63–6
  • Barton A., Jury F., Eyre S., Bowes J., Hinks A., Ward D., et al. Haplotype analysis in simplex families and novel analytic approaches in a case–control cohort reveal no evidence of association of the CTLA‐4 gene with rheumatoid arthritis. Arthritis Rheum 2004; 50: 748–52
  • Hadj‐Kacem H., Kaddour N., Adyel F. Z., Bahloul Z., Ayadi H. HLA‐DQB1 CAR1/CAR2, TNFa IR2/IR4 and CTLA‐4 polymorphisms in Tunisian patients with rheumatoid arthritis and Sjogren's syndrome. Rheumatology 2001; 40: 1370–4
  • Lee Y. H., Choi S. J., Ji J. D., Song G. G. No association of polymorphisms of the CTLA‐4 exon 1 (+49) and promoter (–318) genes with rheumatoid arthritis in the Korean population. Scand J Rheumatol 2002; 31: 266–70
  • Liu M. F., Wang C. R., Chen P. C., Lin T. L. CTLA‐4 gene polymorphism in promoter and exon‐1 regions is not associated with Chinese patients with rheumatoid arthritis. Clin Rheumatol 2004; 23: 180–1
  • Takeuchi F., Kawasugi K., Nabeta H., Mori M., Tanimoto K. CTLA‐4 dimorphisms in Japanese patients with systemic lupus erythematosus. Clin Exp Rheumatol 2003; 21: 527–8
  • Hudson L. L., Rocca K., Song Y. W., Pandey J. P. CTLA‐4 gene polymorphisms in systemic lupus erythematosus: a highly significant association with a determinant in the promoter region. Hum Genet 2002; 111: 452–5

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