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Renal Failure Volume 40, 2018 - Issue 1
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Laboratory Study

Significance of M2 macrophage in tubulointerstitial disease secondary to primary Sjogren's disease

Jun LiWuxi School of Medicine, Jiangnan University, Wuxi, China; ;Department of Nephrology, The Affiliated Hospital of Jiangnan University, Wuxi, China; Correspondence[email protected]
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,
Ya-Fen YuDepartment of Nephrology, The Affiliated Hospital of Jiangnan University, Wuxi, China; View further author information
,
Chang-Hua LiuDepartment of Nephrology, Clinical Medical College, Yangzhou University, Yangzhou, ChinaView further author information
&
Cui-Mei WangDepartment of Nephrology, Clinical Medical College, Yangzhou University, Yangzhou, ChinaView further author information
Pages 634-639 | Received 04 Jun 2017, Accepted 14 Dec 2017, Published online: 05 Nov 2018

References

  • Zhou D, Chen YT, Chen F, et al. Critical involvement of macrophage infiltration in the development of sjogren's syndrome-associated dry eye. Am J Pathol. 2012;181:753–760.
  • Bredberg A, Henriksson G, Larsson A, et al. A role of the macrophage in Sjögen's syndrome? Scand J Rheumatol. 2003;32:255.
  • Greenwell-Wild T, Moutsopoulos NM, Gliozzi M, et al. Chitinases in the salivary glands and circulation of patients with sjogren's syndrome: macrophage harbingers of disease severity. Arthritis Rheum. 2011;63:3103–3115.
  • Ricardo SD, van Goor H, Eddy AA. Macrophage diversity in renal injury and repair. J Clin Invest. 2008;118:3522–3530.
  • Yang N, Isbel NM, Nikolic-Paterson DJ, et al. Local macrophage proliferation in human glomerulonephritis. Kidney Int. 1998;54:143–151.
  • Anders HJ, Ryu M. Renal microenvironments and macrophage phenotypes determine progression or resolution of renal inflammation and fibrosis. Kidney Int. 2011;80:915–925.
  • Stout RD, Jiang C, Matta B, et al. Macrophages sequentially change their functional phenotype in response to changes in microenvironmental influences. J Immunol. 2005;175:342–349.
  • Tateishi Y, Osada-Oka M, Tanaka M, et al. Myeloid HIF-1 attenuates the progression of renal fibrosis in murine obstructive nephropathy. J Pharmacol Sci. 2015;127:181–189.
  • Shiboski SC, Shiboski CH, Criswell L, Sjögren's International Collaborative Clinical Alliance (SICCA) Research Groups, et al. American College of Rheumatology classification criteria for sjogren's syndrome: a data-driven, expert consensus approach in the sjogren's International Collaborative Clinical Alliance cohort. Arthritis Care Res. 2012;64:475–487.
  • Geng Y, Zhang L, Fu B, et al. Mesenchymal stem cells ameliorate rhabdomyolysis-induced acute kidney injury via the activation of M2 macrophages. Stem Cell Res Ther. 2014;5:80.
  • Belliere J, Casemayou A, Ducasse L, et al. Specific macrophage subtypes influence the progression of rhabdomyolysis-induced kidney injury. J Am Soc Nephrol. 2015;26:1363–1377.
  • Vinuesa E, Hotter G, Jung M, et al. Macrophage involvement in the kidney repair phase after ischaemia/reperfusion injury. J Pathol. 2008;214:104–113.
  • Lu J, Cao Q, Zheng D, et al. Discrete functions of M2a and M2c macrophage subsets determine their relative efficacy in treating chronic kidney disease. Kidney Int. 2013;84:745–755.
  • Alagesan S, Griffin MD. Alternatively activated macrophages as therapeutic agents for kidney disease: in vivo stability is a key factor. Kidney Int. 2014;85:730–733.
  • Li J, Liu CH, Xu DL, et al. Clinicopathological significance of CD206-positive macrophages in patients with acute tubulointerstitial disease. Int J Clin Exp Pathol. 2015;8:11386–11392.
  • Li J, Yu YF, Liu CH, et al. Significance of M2 macrophages in glomerulonephritis with crescents. Pathol Res Pract. 2017;213:1215–1220.
  • Song YR, You SJ, Lee YM, et al. Activation of hypoxia-inducible factor attenuates renal injury in rat remnant kidney. Nephrol Dial Transplant. 2010;25:77–85.
  • Conde E, Alegre L, Blanco-Sánchez I, et al. Hypoxia inducible factor 1-alpha (HIF-1 alpha) is induced during reperfusion after renal ischemia and is critical for proximal tubule cell survival. PLoS One. 2012;7:e33258.

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