The molecular and genetic aspects of adolescent girls anomalous uterine bleeding: the role of endothelial dysfunction syndrome

References

• Baranov AA, Kuchma VR, Namazova-Baranova LS. Zdorov’e i razvitie podrostkov Rossii [Health and development of Russian teenagers]. Moscow: NCZD RAMN; 2010:102 (in Russian)
   Google Scholar
• Kokolina VF. Ginekologija detskogo i podrostkovogo vozrasta [Gynecology of childhood and adolescence]. Moscow: Medpraktika; 2006:670 (in Russian)
   Google Scholar
• Uvarova EV. Abnormal uterine bleeding puberty [Reproduktivnoe Zdorov’e Detej I Podrostkov]. 2013;3:73–87
   Google Scholar
• Dan’kova IV, Bashmakova NV, Chistjakova GN. The course of puberty in adolescent girls who were born with the manifestations of intrauterine growth retardation [Reproduktivnoe Zdorov’e Detej I Podrostkov]. 2013;1:44–52 (in Russian)
   Google Scholar
• Lobodina IM. Uterine bleeding puberty (literature review) [Reproduktivnoe Zdorov’e Detej I Podrostkov]. 2007;4:55–71 (in Russian)
   Google Scholar
• (a) Bisadze VO, Makacarija AD, Hizroeva HD, Jashenina EV. Thrombophilia as the most important link in the pathogenesis of complications of pregnancy. [Prakticheskaja medicina]. 2012;5:22–9. (b) Kupferminc MJ. Thrombophilia and pregnancy. Reprod Biol Endocrinol 2003;1:111. doi: 10.1186/1477-7827-1-111
   Google Scholar
• Rey E, Garneau P, David M, et al. Dalteparin for the prevention of recurrence of placental-mediated complications of pregnancy in women without thrombophilia: a pilot randomized controlled trial. J Thromb Haemost 2009;7:58–64
   PubMed Web of Science ®Google Scholar
• Kireeva OV, Sutulina IM. Biochemical markers of endothelial dysfunction and myocardial damage in newborns from mothers with preeclampsia. Mat’i ditja: mat. ХIV Vseros. nauch. foruma [Mother and Child: Materials XIV All-Russia Science Forum]. Moscow, 2013;441–2 (in Russian)
   Google Scholar
• L’vova TJ, Stepanova OI, Postolicyna EA. The formation of vascular endothelial cells line EA.NY926 tubes in the presence of secretory placental tissue products. Mat’i ditja: mat. ХIV Vseros. nauch. Foruma [Mother and Child: Materials XIV All-Russia Science Forum]. Moscow, 2013;111–12 (in Russian)
   Google Scholar
• Clapauch R, Mourão AF, Mecenas AS, et al. Endothelial function and insulin resistance in early postmenopausal women with cardiovascular risk factors: importance of ESR1 and NOS3 polymorphisms. PLoS One 2014;9:e103444. doi:10.1371/journal.pone.0103444
   PubMed Web of Science ®Google Scholar
• Arnal JF, Fontaine C, Billon-Galés A, et al. Estrogens receptors and endothelium. Arterioscler Thromb Vasc Biol 2010;30:1506–12
   PubMed Web of Science ®Google Scholar
• Doshi AA, Ziolo MT, Wang H, et al. Promoter polymorphism of the endothelial nitric oxide synthase gene is associated with reduced mRNA and protein expression in failing human myocardium. J Card Fail 2010;16:314–19
   PubMed Web of Science ®Google Scholar
• Gilchrist M, Shore AC, Benjamin N. Inorganic nitrate and nitrite and control of blood pressure. Cardiovasc Res 2011;15:492–8
   Google Scholar
• Masood D, Roach EC, Beauregard KG, Khalil RA. Impact of sex hormone metabolism on the vascular effects of menopausal hormone therapy in cardiovascular disease. Curr Drug Metab 2010;11:693–714
   PubMed Web of Science ®Google Scholar
• Knowlton AA, Lee AR. Estrogen and the cardiovascular system. Pharmacol Ther 2012;135:54–70
   PubMed Web of Science ®Google Scholar
• Alevizaki M, Saltiki K, Cimponeriu A, et al. Severity of cardiovascular disease in postmenopausal women: associations with common estrogen receptor alpha polymorphic variants. Eur J Endocrinol 2007;156:489–96
   PubMed Web of Science ®Google Scholar
• Heldring N, Pike A, Andersson S, et al. Estrogen receptors: how do they signal and what are their targets. Physiol Rev 2007;87:905–31
   PubMed Web of Science ®Google Scholar
• Bollig A, Miksicek RJ. An estrogen receptor-α splicing variant mediates both positive and negative effects on gene transcription. Mol Endocrinol 2000;14:634–49
   PubMed Web of Science ®Google Scholar
• Chow RW, Handelsman DJ, Ng MK. Minireview: rapid actions of sex steroids in the endothelium. Endocrinology 2010;151:2411–22
   PubMed Web of Science ®Google Scholar
• Entringer S, Buss C, Swanson JM, et al. Fetal programming of body composition, obesity, and metabolic function: the role of intrauterine stress and stress biology. J Nutr Metab 2012;2012:632548. doi: 10.1155/2012/632548
   PubMedGoogle Scholar
• Asif AR, Oellerich M, Armstrong VW, et al. T-786C Polymorphism of the nos-3 gene and the endothelial cell response to fluid shear stress – a proteome analysis. J Prot Res 2009;8:3161–8
   PubMed Web of Science ®Google Scholar
• Erbs S, Möbius-Winkler S, Linke A, et al. Both T-786C and G894T polymorphism of endothelial nitric oxide synthase affect in-vitro endothelium-dependent relaxation of internal mammary artery rings from patients with coronary artery disease. Eur J Cardiovasc Prev Rehabil 2006;13:826–31
   PubMedGoogle Scholar
• Shen C, Chen J, Fan S, et al. Association between the polymorphism of estrogen receptor α and coronary artery disease in a Chinese population. Eur J Intern Med 2012;23:175–8
   PubMed Web of Science ®Google Scholar
• Casey M, MacDonald P. Modulation of endometrial blood flow: regulation of endothelin-1 biosynthesis and degradation in human endometrium. In: Alexander N, d’Arcangues C, eds. Modulation of endometrial blood flow: regulation of endothelin-1 biosynthesis and degradation in human endometrium. Washington (DC): AAAS Press; 1992. p. 209
   Google Scholar
• Cao Y, Linden P, Shima D, et al. In vivo angiogenic activity and hypoxia induction of heterodimers of placenta growth factor/vascular endothelial growth factor. J Clin Invest 1996;98:2507–11
   PubMed Web of Science ®Google Scholar
• Zil’ber MJ, Grebneva OS, Cyganenko SV. Status of the receptor apparatus of the endometrium in acute placental insufficiency in history. Mat’i ditja: mat. ХIV Vseros. nauch. foruma [Mother and Child: Materials XIV All-Russia Science Forum]. Moscow, 2013:69–70 (in Russian)
   Google Scholar
• Lockwood CJ, Nemerson Y, Guller S, et al. Decidualized human endometrial stromal cells mediate hemostasis, angiogenesis, and abnormal uterine bleeding. Reprod Sci Feb 2009;16:162–70
   PubMed Web of Science ®Google Scholar
• Nofer JR. Estrogens and atherosclerosis: insights from animal models and cell systems. J Mol Endocrinol 2012;48:R13–29
   Google Scholar
• Lockwood CJ. Mechanisms of normal and abnormal endometrial bleeding. Menopause 2011;18:408–11
   PubMed Web of Science ®Google Scholar