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Letters to the Editor

Association of GSTP1 inactivating polymorphism with acute myeloid leukemia and its specific chromosomal abnormalities

, , , , , , & show all
Pages 2505-2507 | Received 04 Jan 2017, Accepted 05 Feb 2017, Published online: 02 Mar 2017

References

  • Hengstler JG, Arand M, Herrero ME, et al. Polymorphisms of N-acetyltransferases, glutathione S-transferases, microsomal epoxide hydrolase, and sulfotransferases: influence on cancer susceptibility. Recent Results Cancer Res. 1998;154:47.
  • Perera F. Molecular epidemiology: insights into cancer susceptibility, risk assessment, and prevention. J Natl Cancer Inst. 1996;88:496.
  • Borde-Chiché P, Diederich M, Morceau F, et al. Regulation of transcription of the glutathione S-transferase P1 gene by methylation of the minimal promoter in human leukemia cells. Biochem Pharmacol. 2001;61:605–612.
  • Ali-Osman F, Akande O, Antoun G, et al. Molecular cloning, characterization, and expression in Escherichia coli of full-length cDNAs of three human glutathione S-transferase Pi gene variants. Evidence for differential catalytic activity of the encoded proteins. J Biol Chem. 1997;272:10004–10012.
  • Watson MA, Stewart RK, Smith GBJ, et al. Human glutathione S-transferase P1 polymorphisms: relationships to lung tissue enzyme activity and population frequency distribution. Carcinogenesis. 1998;19:275.
  • Zachaki S, Stavropoulou C, Kalomoiraki M, et al. Association of A(313)G glutathione S-transferase P1 germline polymorphism with susceptibility to de novo myelodysplastic syndrome. Leuk Lymphoma. 2013;54:1756–1761.
  • Bănescu C, Iancu M, Trifa AP, et al. From six gene polymorphisms of the antioxidant system, only GPX Pro198Leu and GSTP1 Ile105Val modulate the risk of acute myeloid leukemia. Oxid Med Cell Longev. 2016;2016:2536705.
  • Dunna NR, Vuree S, Kagita S, et al. Association of GSTP1 gene (I105V) polymorphism with acute leukaemia. J Genet. 2012;1791:60–63.
  • Voso MT, Hohaus S, Guidi F, et al. Prognostic role of glutathione S-transferase polymorphisms in acute myeloid leukemia. Leukemia. 2008;22:1685–1691.
  • Nasr AS, Sami RM, Ibrahim NY, et al. Glutathione S transferase (GSTP 1, GSTM 1, and GSTT 1) gene polymorphisms in Egyptian patients with acute myeloid leukemia. Indian J Cancer. 2015;52:490–495.
  • Allan JM, Wild CP, Rollinson S, et al. Polymorphism in glutathione S-transferase P1 is associated with susceptibility to chemotherapy-induced leukemia. Proc Natl Acad Sci USA. 2001;98:11592–11597.
  • Tang ZH, Zhang C, Cheng P, et al. Glutathione-S-transferase polymorphisms (GSTM1, GSTT1 and GSTP1) and acute leukemia risk in Asians: a meta-analysis. Asian Pac J Cancer Prev. 2014;15:2075–2081.
  • He HR, You HS, Sun JY, et al. Glutathione S-transferase gene polymorphisms and susceptibility to acute myeloid leukemia: meta-analyses. Jpn J Clin Oncol. 2014;44:1070–1081.
  • Mauritzson N, Albin M, Rylander L, et al. Pooled analysis of clinical and cytogenetic features in treatment-related and de novo adult acute myeloid leukemia and myelodysplastic syndromes based on a consecutive series of 761 patients analyzed 1976-1993 and on 5098 unselected cases reported in the literature 1974-2001. Leukemia. 2002;16:2366–2378.
  • Kim SY, Choi JK, Cho YH, et al. Chromosomal aberrations in workers exposed to low levels of benzene: association with genetic polymorphisms. Pharmacogenetics. 2004;14:453–463.

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