References
- Smolen JS, Aletaha D, Steiner G. Does damage cause inflammation? Revisiting the link between joint damage and inflammation. Ann Rheum Dis. 2009;68(2):159–162.
- Carmona L, Villaverde V, Hernández‐García C, et al. The prevalence of rheumatoid arthritis in the general population of Spain. Rheumatology (Oxford). 2002;41(1):88–95.
- Datta S, Kundu S, Ghosh P, et al. Correlation of oxidant status with oxidative tissue damage in patients with rheumatoid arthritis. Clin Rheumatol. 2014;33(11):1557–1564.
- Pradhan A, Bagchi A, De S, et al. Role of redox imbalance and cytokines in mediating oxidative damage and disease progression of patients with rheumatoid arthritis. Free Radic Res. 2019;53(7):768–779.
- McInnes IB, Schett G. The pathogenesis of rheumatoid arthritis. N Engl J Med. 2011;365(23):2205–2219.
- Geng S, Matsushima H, Okamoto T, et al. Reciprocal regulation of development of neutrophil-dendritic cell hybrids in mice by IL-4 and interferon-gamma. PLoS One. 2013;8(11):e82929.
- Chakravarti A, Rusu D, Flamand N, et al. Reprogramming of a subpopulation of human blood neutrophils by prolonged exposure to cytokines. Lab Invest. 2009;89(10):1084–1099.
- Matsushima H, Geng S, Lu R, et al. Neutrophil differentiation into a unique hybrid population exhibiting dual phenotype and functionality of neutrophils and dendritic cells. Blood J Am Soc Hematol. 2013;121(10):1677–1689.
- Iking-Konert C, Ostendorf B, Sander O, et al. Transdifferentiation of polymorphonuclear neutrophils to dendritic-like cells at the site of inflammation in rheumatoid arthritis: evidence for activation by T cells. Ann Rheum Dis. 2005;64(10):1436–1442.
- Chitnis D, Dickerson C, Munster AM, et al. Inhibition of apoptosis in polymorphonuclear neutrophils from burn patients. J Leukoc Biol. 1996;59(6):835–839.
- Costa S, Bevilacqua D, Cassatella MA, et al. Recent advances on the crosstalk between neutrophils and B or T lymphocytes. Immunology. 2019;156(1):23–32.
- van der Linden MPM, Batstra MR, Bakker-Jonges LE, et al. Toward a data-driven evaluation of the 2010 American college of rheumatology/European league against rheumatism criteria for rheumatoid arthritis: is it sensible to look at levels of rheumatoid factor? Arthritis Rheum. 2011;63(5):1190–1199.
- Kokkonen H, Söderström I, Rocklöv J, et al. Upregulation of cytokines and chemokines predates the onset of rheumatoid arthritis. Arthritis Rheum. 2010;62(2):383–391.
- Takashima A, Yao Y. Neutrophil plasticity: acquisition of phenotype and functionality of antigen-presenting cell. J Leukoc Biol. 2015;98(4):489–496.
- Kundu S, Bala A, Ghosh P, et al. Attenuation of oxidative stress by allylpyrocatechol in synovial cellular infiltrate of patients with rheumatoid arthritis. Free Radic Res. 2011;45(5):518–526.
- Kundu S, Ghosh P, Datta S, et al. Oxidative stress as a potential biomarker for determining disease activity in patients with rheumatoid arthritis. Free Radic Res. 2012;46(12):1482–1489.
- Jacobson EC, Jain L, Vickers MH, et al. TNF-α differentially regulates cell cycle genes in promyelocytic and granulocytic HL-60/S4 cells. G3. G3 (Bethesda). 2019;9(8):2775–2786.
- Gosselin EJ, Wardwell K, Rigby WF, et al. Induction of MHC class II on human polymorphonuclear neutrophils by granulocyte/macrophage colony-stimulating factor, IFN-gamma, and IL-3. J Immunol. 1993;151(3):1482–1490.
- Benna JE, Dang PM, Andrieu V, et al. P40phox associates with the neutrophil triton X-100-insoluble cytoskeletal fraction and PMA-activated membrane skeleton: a comparative study with P67phox and P47phox. J Leukoc Biol. 1999;66(6):1014–1020.
- Dewas C, Dang PM, Gougerot-Pocidalo MA, et al. TNF-alpha induces phosphorylation of p47(phox) in human neutrophils: partial phosphorylation of p47phox is a common event of priming of human neutrophils by TNF-alpha and granulocyte-macrophage colony-stimulating factor. J Immunol. 2003;171(8):4392–4398.
- Ellis JH, Burden MN, Vinogradov DV, et al. Interactions of CD80 and CD86 with CD28 and CTLA4. J Immunol. 1996;156(8):2700–2709.
- Woodfin A, Beyrau M, Voisin MB, et al. ICAM-1-expressing neutrophils exhibit enhanced effector functions in murine models of endotoxemia. Blood J Am Soc Hematol. 2016;127(7):898–907.
- De S, Manna A, Kundu S, et al. Allylpyrocatechol attenuates Collagen-Induced Arthritis via Attenuation of Oxidative Stress Secondary to Modulation of the MAPK, JAK/STAT, and Nrf2/HO-1 Pathways. J Pharmacol Exp Ther. 2017;360(2):249–259.
- Takeshita M, Suzuki K, Kondo Y, et al. Multi-dimensional analysis identified rheumatoid arthritis-driving pathway in human T cell. Ann Rheum Dis. 2019;78(10):1346–1356.
- Lin A, Loré K. Granulocytes: new members of the antigen-presenting cell family. Front Immunol. 2017;8:1781.