Apolipoprotein A-II accelerates reactive AA amyloidosis

Mu YangDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan,

Yingye LiuDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan,

Lin LiDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan,

Hiroki MiyaharaDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan,

Xin DingDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan,

Jian DaiDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan,

Jinko SawashitaDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan, ;Department of Biological Sciences for Intractable Neurological Diseases, Shinshu University, Matsumoto, Japan, and

Masayuki MoriDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan, ;Department of Advanced Medicine for Health Promotion, IBS-ICCER, Shinshu University, Matsumoto, Japan

Keiichi HiguchiDepartment of Aging Biology, Institute of Pathogenesis and Disease Prevention, Shinshu University Graduate School of Medicine, Matsumoto, Japan, ;Department of Biological Sciences for Intractable Neurological Diseases, Shinshu University, Matsumoto, Japan, and Correspondence[email protected]

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References

Yan J, Fu X, Ge F, Zhang B, Yao J, Zhang H, Qian J, et al. Cross-seeding and cross-competition in mouse apolipoprotein A-II amyloid fibrils and protein A amyloid fibrils. Am J Pathol 2007;171:172–80
PubMed Web of Science ®Google Scholar
Wang Y, Sawashita J, Qian J, Zhang B, Fu X, Tian G, Chen L, et al. ApoA-I deficiency in mice is associated with redistribution of apoA-II and aggravated AApoAII amyloidosis. J Lipid Res 2011;52:1461–70
PubMed Web of Science ®Google Scholar
Thompson PA, Berbée JF, Rensen PC, Kitchens RL. Apolipoprotein A-II augments monocyte responses to LPS by suppressing the inhibitory activity of LPS-binding protein. Innate Immun 2008;14:365–74
PubMed Web of Science ®Google Scholar

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