Red blood cell folate and benign prostatic hyperplasia: results from the NHANES 2001-2008

References

• Foo KT. What is a disease? What is the disease clinical benign prostatic hyperplasia (BPH)? World J Urol. 2019;37(7):1293–1296. doi: 10.1007/s00345-019-02691-0.
   PubMed Web of Science ®Google Scholar
• Langan RC. Benign prostatic hyperplasia. Prim Care. 2019;46(2):223–232. doi: 10.1016/j.pop.2019.02.003.
   PubMed Web of Science ®Google Scholar
• Zhu C, Wang D-Q, Zi H, et al. Epidemiological trends of urinary tract infections, urolithiasis and benign prostatic hyperplasia in 203 countries and territories from 1990 to 2019. Mil Med Res. 2021;8(1):64. doi: 10.1186/s40779-021-00359-8.
   PubMed Web of Science ®Google Scholar
• Ng M, Baradhi KM. Benign Prostatic Hyperplasia. In StatPearls; StatPearls Publishing: treasure Island (FL), 2023.
   Google Scholar
• Launer BM, McVary KT, Ricke WA, et al. The rising worldwide impact of benign prostatic hyperplasia. BJU Int. 2021;127(6):722–728. doi: 10.1111/bju.15286.
   PubMed Web of Science ®Google Scholar
• Yang L, Liu Z, Peng Z, et al. Exposure to di-2-ethylhexyl phthalate and benign prostatic hyperplasia, NHANES 2001-2008. Front Endocrinol. 2021;12:804457. doi: 10.3389/fendo.2021.804457.
   PubMed Web of Science ®Google Scholar
• Devlin CM, Simms MS, Maitland NJ. Benign prostatichyperplasia - What do We know? BJU Int. 2021;127(4):389–399. doi: 10.1111/bju.15229.
   PubMed Web of Science ®Google Scholar
• S, S, Ea P, I K, Wc, et al. Intakes of energy and macronutrients and the risk of benign prostatic hyperplasia. A f Clin Nutr. 2002;75(4):689–697. doi: 10.1093/ajcn/75.4.689.
   Google Scholar
• Das K, Buchholz N. Benign prostate hyperplasia and nutrition. Clin Nutr ESPEN. 2019;33:5–11. doi: 10.1016/j.clnesp.2019.07.015.
   PubMed Web of Science ®Google Scholar
• Zhou H, Xu M, Pan Y, et al. The association between several serum micronutrients and benign prostatic hyperplasia: results from NHANES 2003-2006. Prostate. 2024;84(2):212–220. doi: 10.1002/pros.24641.
   PubMed Web of Science ®Google Scholar
• Shulpekova Y, Nechaev V, Kardasheva S, et al. The concept of folic acid in health and disease. Molecules. 2021;26(12):3731. doi: 10.3390/molecules26123731.
   PubMed Web of Science ®Google Scholar
• Wald NJ, Hoffbrand AV. Mandatory UK folic acid fortification. Lancet. 2021;398(10315):1961–1962. doi: 10.1016/S0140-6736(21)02447-8.
   PubMed Web of Science ®Google Scholar
• Crider KS, Qi YP, Yeung LF, et al. Folic acid and the prevention of birth defects: 30 years of opportunity and controversies. Annu Rev- Nutr. 2022;42(1):423–452. doi: 10.1146/annurev-nutr-043020-091647.
   PubMedGoogle Scholar
• Benavides-Lara A, Fernández-Sánchez O, Barboza-Argüello M, et al. Integrated surveillance strategy to support the prevention of neural tube defects through food fortification with folic acid: the experience of Costa Rica. Childs Nerv Syst. 2023;39(7):1743–1754. doi: 10.1007/s00381-023-05837-z.
   PubMed Web of Science ®Google Scholar
• Field MS, Stover PJ. Safety of folic acid. Ann N Y Acad Sci. 2018;1414(1):59–71. doi: 10.1111/nyas.13499.
   PubMed Web of Science ®Google Scholar
• Patel KR, Sobczyńska-Malefora A. The adverse effects of an excessive folic acid intake. Eur J Clin Nutr. 2017;71(2):159–163. doi: 10.1038/ejcn.2016.194.
   PubMed Web of Science ®Google Scholar
• Selhub J, Rosenberg IH. Excessive folic acid intake and relation to adverse health outcome. Biochimie. 2016;126:71–78. doi: 10.1016/j.biochi.2016.04.010.
   PubMed Web of Science ®Google Scholar
• Zhu J, Jia Y-L, Luo Y-W, et al. Effect of maternal folic acid supplementation on prostatitis risk in the rat offspring. Int Urol Nephrol. 2018;50(11):1963–1973. doi: 10.1007/s11255-018-1969-8.
   PubMed Web of Science ®Google Scholar
• Zhou L, Wen X, Peng Y, et al. Red blood cell folate and severe abdominal aortic calcification: results from the NHANES 2013-2014. Nutr Metab Cardiovasc Dis. 2021;31(1):186–192. doi: 10.1016/j.numecd.2020.08.020.
   PubMed Web of Science ®Google Scholar
• Inamura S, Ito H, Shinagawa T, et al. Serum C-reactive protein level is not associated with prostatic inflammation but with overactive detrusor in patients with benign prostatic hyperplasia. Neurourol Urodyn. 2019;38(6):1728–1736. doi: 10.1002/nau.24051.
   PubMed Web of Science ®Google Scholar
• Wang Y-B, Yang L, Deng Y-Q, et al. Causal relationship between obesity, lifestyle factors and risk of benign prostatic hyperplasia: a univariable and multivariable mendelian randomization study. J Transl Med. 2022;20(1):495. doi: 10.1186/s12967-022-03722-y.
   PubMed Web of Science ®Google Scholar
• Mentch SJ, Locasale JW. One-carbon metabolism and epigenetics: understanding the specificity. Ann N Y Acad Sci. 2016;1363(1):91–98. doi: 10.1111/nyas.12956.
   PubMedGoogle Scholar
• Malinowska AM, Schmidt M, Kok DE, et al. Ex vivo folate production by fecal bacteria does not predict human blood folate status: associations between dietary patterns, gut microbiota, and folate metabolism. Food Res Int. 2022;156:111290. doi: 10.1016/j.foodres.2022.111290.88
   PubMed Web of Science ®Google Scholar
• Viswanathan M, Treiman KA, Kish-Doto J, et al. Folic acid supplementation for the prevention of neural tube defects: an updated evidence report and systematic review for the US preventive services task force. JAMA. 2017;317(2):190–203. doi: 10.1001/jama.2016.19193.
   PubMed Web of Science ®Google Scholar
• Chen M-Y, Rose CE, Qi YP, et al. Defining the plasma folate concentration associated with the red blood cell folate concentration threshold for optimal neural tube defects prevention: a population-based, randomized trial of folic acid supplementation. Am J Clin Nutr. 2019;109(5):1452–1461. doi: 10.1093/ajcn/nqz027.
   PubMed Web of Science ®Google Scholar
• Dietary Intake of Folate and Assessment of the Folate Deficiency Prevalence in Slovenia Using Serum Biomarkers - PubMed. https://pubmed.ncbi.nlm.nih.gov/34836112/. (accessed 2023-07-20).
   Google Scholar
• Hiraoka M, Kagawa Y. Genetic polymorphisms and folate status. Congenit Anom. 2017;57(5):142–149. doi: 10.1111/cga.12232.
   PubMed Web of Science ®Google Scholar
• Ledowsky C, Mahimbo A, Scarf V, et al. Women taking a folic acid supplement in countries with mandatory food fortification programs may be exceeding the upper tolerable limit of folic acid: a systematic review. Nutrients. 2022;14(13):2715. doi: 10.3390/nu14132715.
   PubMed Web of Science ®Google Scholar
• Leclerc D, Jelinek J, Christensen KE, et al. High folic acid intake increases methylation-dependent expression of lsr and dysregulates hepatic cholesterol homeostasis. J Nutr Biochem. 2021;88:108554. doi: 10.1016/j.jnutbio.2020.108554.
   PubMed Web of Science ®Google Scholar
• Pieroth R, Paver S, Day S, et al. Folate and its impact on cancer risk. Curr Nutr Rep. 2018;7(3):70–84. doi: 10.1007/s13668-018-0237-y.
   PubMedGoogle Scholar
• Rycyna KJ, Bacich DJ, O Keefe DS. Divergence between dietary folate intake and concentrations in the serum and red blood cells of aging males in the United States. Clin Nutr. 2016;35(4):928–934. doi: 10.1016/j.clnu.2015.07.002.
   PubMed Web of Science ®Google Scholar
• Awwad HM, Ohlmann C-H, Stoeckle M, et al. Serum concentrations of folate vitamers in patients with a newly diagnosed prostate cancer or hyperplasia. Clin Biochem. 2018;56:41–46. doi: 10.1016/j.clinbiochem.2018.04.011.
   PubMed Web of Science ®Google Scholar
• Marosi K, Agota A, Végh V, et al. [The role of homocysteine and methylenetetrahydrofolate reductase, methionine synthase, methionine synthase reductase polymorphisms in the development of cardiovascular diseases and hypertension]. Orv Hetil. 2012;153(12):445–453. doi: 10.1556/OH.2012.29326.
   PubMedGoogle Scholar
• Tavani A, Longoni E, Bosetti C, et al. Intake of selected micronutrients and the risk of surgically treated benign prostatic hyperplasia: a case-control study from Italy. Eur Urol. 2006;50(3):549–554. doi: 10.1016/j.eururo.2005.11.027.
   PubMed Web of Science ®Google Scholar