References
- McInnes IB, Schett G. Cytokines in the pathogenesis of rheumatoid arthritis. Nat Rev Immunol. 2007;7(6):429–42.
- Inoue A, Matsumoto I, Tanaka Y, Umeda N, Tanaka Y, Mihara M, et al. Murine tumor necrosis factor α-induced adipose-related protein (tumor necrosis factor α-induced protein 9) deficiency leads to arthritis via interleukin-6 overproduction with enhanced NF-κB, STAT-3 signaling, and dysregulated apoptosis of macrophages. Arthritis Rheum. 2012;64(12):3877–85.
- Inoue A, Matsumoto I, Tanaka Y, Umeda N, Takai C, Kawaguchi H, et al. TIARP attenuates autoantibody-mediated arthritis via the suppression of neutrophil migration by reducing CXCL2/CXCR2 and IL-6 expression. Sci Rep. 2016;6(1):38684.
- Korkmaz CG, Korkmaz KS, Kurys P, Elbi C, Wang L, Klokk TI, et al. Molecular cloning and characterization of STAMP2, an androgen-regulated six transmembrane protein that is overexpressed in prostate cancer. Oncogene 2005;24(31):4934–45.
- Takai C, Matsumoto I, Inoue A, Umeda N, Tanaka Y, Kurashima Y, et al. Specific overexpression of tumour necrosis factor-α-induced protein (TNFAIP)9 in CD14(+) CD16(–) monocytes in patients with rheumatoid arthritis: comparative analysis with TNFAIP3. Clin Exp Immunol. 2015;180(3):458–66.
- Ebe H, Matsumoto I, Kawaguchi H, Kurata I, Tanaka Y, Inoue A, et al. Clinical and functional significance of STEAP4-splice variant in CD14+ monocytes in patients with rheumatoid arthritis. Clin Exp Immunol. 2018;191(3):338–48.
- Tanaka Y, Matsumoto I, Iwanami K, Inoue A, Minami R, Umeda N, et al. Six-transmembrane epithelial antigen of prostate4 (STEAP4) is a tumor necrosis factor alpha-induced protein that regulates IL-6, IL-8, and cell proliferation in synovium from patients with rheumatoid arthritis. Mod Rheumatol. 2012;22(1):128–36.
- Xue X, Bredell BX, Anderson ER, Martin A, Mays C, Nagao-Kitamoto H, et al. Quantitative proteomics identifies STEAP4 as a critical regulator of mitochondrial dysfunction linking inflammation and colon cancer. Proc Natl Acad Sci USA. 2017;114(45):E9608–17.
- Stamm S, Ben-Ari S, Rafalska I, Tang Y, Zhang Z, Toiber D, et al. Function of alternative splicing. Gene. 2005;344:1–20.
- Comabella M. Neuroimmunology: B cells and variant BAFF in autoimmune disease. Nat Rev Neurol. 2017;13(8):453–4.
- Roescher N, Vosters JL, Alsaleh G, Dreyfus P, Jacques S, Chiocchia G, et al. Targeting the splicing of mRNA in autoimmune diseases: BAFF inhibition in Sjögren’s syndrome as a proof of concept. Mol Ther. 2014;22(4):821–7.
- Kochi Y, Kamatani Y, Kondo Y, Suzuki A, Kawakami E, Hiwa R, et al. Splicing variant of WDFY4 augments MDA5 signalling and the risk of clinically amyopathic dermatomyositis. Ann Rheum Dis. 2018;77(4):602–11.
- Kim JY, Yoon HK, Song ST, Park SR, Shim SC. Expression of activation-induced cytidine deaminase splicing variants in patients with ankylosing spondylitis. Autoimmunity. 2017;50(8):435–40.