Maternal intravascular inflammation in preterm premature rupture of membranes

References

• Mercer BM, Lewis R. Preterm labor and preterm premature rupture of the membranes. Diagnosis and management. Infect Dis Clin North Am 1997;11:177–201
   PubMed Web of Science ®Google Scholar
• Gomez R, Ghezzi F, Romero R, et at. Premature labor and intra-amniotic infection: clinical aspects and role of the cytokines in diagnosis and pathophysiology. Clin Perinatol 1995;22:281–342
   PubMed Web of Science ®Google Scholar
• Parry S, Strauss FJ. Premature rupture of the fetal membranes.N Engl J Med 1998;338:663–70
   PubMed Web of Science ®Google Scholar
• Vitzileos AM. Antepartum surveillance in preterm rupture of membranes. J Perinat Med 1996;24:319–26
   Google Scholar
• Romero R, Gomez R, Ghezzi F, et at. A fetal systemic inflam-matory response is followed by the spontaneous onset of preterm parturition. Am J Obstet Gynecol 1998;179:186–93
   PubMed Web of Science ®Google Scholar
• Dudley D, Trautman M, Araneo B, et at. Decidual cell bio-synthesis of interleukin-6: regulation by inflammatory cytokines.J Clin EndocrinolMetab 1992;74:884–9
   PubMed Web of Science ®Google Scholar
• Fortunato SJ, Menon R, Swan KF. Expression of TNF-alpha and TNFR p55 in cultured amniochorion. Am J Reprod Immunol 1994;32: 188–93
   Google Scholar
• Fortunato SJ, Menon RP, Swan KF, et at. Inflammatory cyto-kine (interleukins 1, 6 and tumor necrosis factor-a) release from cultured human fetal membranes in response to endo-toxic lipopolysaccharide mirrors amniotic fluid concentra-tions. Am J Obstet Gynecol 1996;174:1855–62
   PubMed Web of Science ®Google Scholar
• Parry S, Angus SR, Hsu CD, et at. Matrix metalloproteinase-8 is a marker for intraamniotic infection. Sac Gynecol Invest 2000;7:abstract 829
   Google Scholar
• Watari M, Watari H, DiSanto ME, et at. Pro-inflammatory cytokines induce expression of matrix-metabolizing enzymes in human cervical smooth muscle cells. Am J Pathol 1999;154: 1755–62
   PubMed Web of Science ®Google Scholar
• Buhimschi IA, Kramer WB, Buhimschi CS, et at. Reduction-oxidation (redox) state regulation of matrix metalloproteinase activity in human fetal membranes. Am J Obstet Gynecol 2000;182:458–64
   PubMed Web of Science ®Google Scholar
• Fortunato SJ, Menon R, Bryant C, et at. Programmed cell death ( ap op tos is) as a possible pathway to metalloproteinase activa-tion and fetal membrane degradation in premature rupture of membranes. Am J Obstet Gynecol 2000;182:1468–76
   PubMed Web of Science ®Google Scholar
• Barclay AN, Brown MH, Law SKA, et at. The Leukocyte Antigen Facts Book, 2nd edn. San Diego: Academic Press, 1997
   Google Scholar
• McCarthy DA, Macey MG. Novel anticoagulants for flow cytometric analysis of live leukocytes in whole blood. Cytometry 1996;23:196–204
   PubMedGoogle Scholar
• McCarthy DA, Macey MG. A simple flow cytometric proce-dure for the determination of surface antigens on unfixed leukocytes in whole blood. J Immunol Meth 1993;163:155–63
   PubMed Web of Science ®Google Scholar
• Himmelfarb J, Hakim RM, Holbrook DO, et at. Detection of granulocyte reactive oxygen species formation in whole blood using flow cytometry. Cytometry 1992;13:83–9
   PubMedGoogle Scholar
• Gervasi MT, Chaiworapongsa T, Pacora P, et at. Phenotypic and metabolic characteristics of monocytes and granulocytes in preeclampsia. Am J Obstet Gynecol 2001;185:792–7
   PubMed Web of Science ®Google Scholar
• Gervasi MT, Chaiworapongsa T, Naccasha N, et at. Differen-tial expressionof the cell surface receptor (CD95) for the death cell factor Fas-Ligand in p ree clamps ia and maternal infection. Am J Obstet Gynecol 2001;184:abstract0026
   Google Scholar
• Naccasha N, Gervasi MT, Chaiworapongsa T, et at. Pheno-typic and metabolic characteristics of monocytes and granulo-cytes in normal pregnancy and maternal infection. Am J Obstet Gynecol 2001;185:1118–23
   PubMed Web of Science ®Google Scholar
• Gervasi MT, Chaiworapongsa T, Naccasha N, et at. Pheno-typic and metabolic characteristics of maternal monocytes and granulocytes in preterm labor with intact membranes. Am J Obstet Gynecol 2001;185:1124–9
   PubMed Web of Science ®Google Scholar
• Rosenbloom AJ, Pinsky MR, Napolitano C, et at. Suppression of cytokine-mediated 02-integrin activation on circulating neutrophils in critically ill patients. JLeukocBiol 1999;65:83–9
   Google Scholar
• Diamond MS, Springer TA. A subpopulation of MAC-1 (CD1 lb/CD18) molecules mediates neutrophil adhesion to ICAM-1 and fibrinogen. J Cell Biol 1993;120:545–6
   PubMed Web of Science ®Google Scholar
• Wright SD, Ramos RA, Hermanowski-Vosatka A, et at. Activation of the adhesive capacity of CR3 on neutrophils by endotox in: dependence on lipopolysaccharideb inding protein and CD14. J Exp Med 1991;173: 1281–6
   Google Scholar
• Ferrero E, J iao D, Tsuberi BZ, et at. Transgenic mice expressing human CD14 are hypersensitive to lipopolysaccharide. Proc Natl Acad Sci USA 1993;90:2380–4
   PubMed Web of Science ®Google Scholar
• Haziot A, Ferrero E, Kontgen F, et at. Resistance to endotoxin shock and reduced dissemination of gram-negative bacteria in CD14 -deficient mice. Immunity 1996;4:407–14
   PubMed Web of Science ®Google Scholar