References
- Wang HJ, Varner A, AbouShwareb T, et al. Ischemia/reperfusion-induced renal failure in rats as a model for evaluating cell therapies. Ren Fail. 2012;34:1324–1332
- Liu X, Chen H, Zhan B, et al. Attenuation of reperfusion injury by renal ischemic postconditioning: The role of NO. Biochem Biophys Res Commun. 2007;359:628–634
- Chen H, Xing B, Liu X, et al. Ischemic postconditioning inhibits apoptosis after renal ischemia/reperfusion injury in rat. Transpl Int. 2008;21:364–371
- Weng X, Shen H, Kuang Y, et al. Ischemic postconditioning inhibits the renal fibrosis induced by ischemia-reperfusion injury in rats. Urology. 2012;80:484e1–484e7
- Jiang B, Liu X, Chen H, et al. Ischemic postconditioning attenuates renal ischemic/reperfusion injury in mongrel dogs. Urology. 2010;76:1519e1–1519e7
- Jiang B, Chen Q, Liu X, et al. Ischemic postconditioning protects renal function after 24 hours of cold preservation in a canine autotransplantation model. Transplant Proc. 2012;44:1776–1781
- Wang W, Tang T, Zhang P, et al. Postconditioning attenuates renal ischemia-reperfusion injury by preventing DAF down-regulation. J Urol. 2010;183:2424–2431
- Zhang WL, Zhao YL, Liu XM, et al. Protective role of mitochondrial K-ATP channel and mitochondrial membrane transport pore in rat kidney ischemic postconditioning. Chin Med J (Engl). 2011;124:2191–2195
- Serviddio G, Romano AD, Gesualdo L, et al. Postconditioning is an effective strategy to reduce renal ischemia/reperfusion injury. Nephrol Dial Transplant. 2008;23:1504–1512
- Eldaif SM, Deneve JA, Wang NP, et al. Attenuation of renal ischemia-reperfusion injury by postconditioning involves adenosine receptor and protein kinase C activation. Transpl Int. 2010;23:217–226
- Yun Y, Duan WG, Chen P, et al. Ischemic postconditioning modified renal oxidative stress and lipid peroxidation caused by ischemic reperfusion injury in rats. Transplant Proc. 2009;41:3597–3602
- Yun Y, Duan WG, Chen P, et al. Down-regulation of cyclooxygenase-2 is involved in ischemic postconditioning protection against renal ischemia reperfusion injury in rats. Transplant Proc. 2009;41:3585–3589
- Van den Akker EK, Hesselink DA, Manintveld OC, et al. Ischemic postconditioning in human DCD kidney transplantation is feasible and appears safe. Transpl Int. 2014;27:226–234
- Jablonski P, Howden BO, Rae DA, et al. An experimental model for assessment of renal recovery from warm ischemia. Transplantation. 1983;35:198–204
- Matsumori Y, Hong SM, Aoyama K, et al. Hsp70 overexpression sequesters AIF and reduces neonatal hypoxic/ischemic brain injury. J Cereb Blood Flow Metab. 2005;25:899–910
- Staat P, Rioufol G, Piot C, et al. Postconditioning the human heart. Circulation. 2005;112:2143–2138
- Hausenloy DJ, Yellon DM. The therapeutic potential of ischemic conditioning: An update. Nat Rev Cardiol. 2011;8:619–629
- Xing B, Chen H, Zhang M, et al. Ischemic postconditioning inhibits apoptosis after focal cerebral ischemia/reperfusion injury in the rat. Stroke. 2008;39:2362–2369
- Boengler K, Buechert A, Heinen Y, et al. Cardioprotection by ischemic postconditioning is lost in aged and STAT3-deficient mice. Circ Res. 2008;102:131–135
- Przyklenk K, Maynard M, Darling CE, et al. Aging mouse hearts are refractory to infarct size reduction with post-conditioning. J Am Coll Cardiol. 2008;51:1393–1398
- Kusaka J, Koga H, Hagiwara S, et al. Age-dependent responses to renal ischemia-reperfusion injury. J Surg Res. 2012;172:153–158
- Qiao X, Chen X, Wu D, et al. Mitochondrial pathway is responsible for aging-related increase of tubular cell apoptosis in renal ischemia/reperfusion injury. J Gerontol A Biol Sci Med Sci. 2005;60:830–839
- Xu X, Fan M, He X, et al. Aging aggravates long-term renal ischemia-reperfusion injury in a rat mode. J Surg Res. 2014;187:289–296
- Padanilam BJ. Cell death induced by acute renal injury: A perspective on the contributions of apoptosis and necrosis. Am J Physiol Renal Physiol. 2003;284:608–627
- Shimizu S, Narita M, Tsujimoto Y, et al. Bcl-2 family pro-teins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC. Nature. 1999;399:483–487
- Li P, Nijhawan D, Budihardio I, et al. Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade. Cell. 1997;91:479–489