Oxidant generation capacity of source-apportioned PM2.5

References

• Chen LC, Hwang JS. 2005. Effects of subchronic exposures to concentrated ambient particles (CAPs) in mice. IV. Characterization of acute and chronic effects of ambient air fine particulate matter exposures on heart-rate variability. Inhal Toxicol 17:209–216.
   PubMed Web of Science ®Google Scholar
• Chen LC, Hwang JS, Lall R, Thurston G, Lippmann M. 2010. Alteration of cardiac function in ApoE−/− mice by subchronic urban and regional inhalation exposure to concentrated ambient PM 2.5. Inhal Toxicol 22:580–592.
   PubMed Web of Science ®Google Scholar
• Dominici F, McDermott A, Daniels M, Zeger SL, Samet JM. 2003. Special report. Boston, MA: Health Effects Institute. Mortality among residents of 90 cities. In: Revised Analyses of Time-Series Studies of Air Pollution and Health. Available: http://pubs.healtheffects.org/view.php?id=4 [Accessed 17 August 2010].
   Google Scholar
• Dominici F, Peng RD, Ebisu K, Zeger SL, Samet JM, Bell ML. 2007. Does the effect of PM10 on mortality depend on PM nickel and vanadium content? A reanalysis of the NMMAPS data. Environ Health Perspect 115:1701–1703.
   PubMed Web of Science ®Google Scholar
• Fanning E, Froines J, Utell M, Lippmann M, Oberdorster G, Godleski J, Larson T. 2007. Accomplishments of the Particulate Matter (PM) Centers (1999–2005) and the Role of Interdisciplinary Center-Based. Report to EPA. Available at: http://www.epa.gov/ncer/science/pm/documents/11543.pdf (Accessed June 17, 2010).
   Google Scholar
• Fujii T, Hayashi S, Hogg JC, Mukae H, Suwa T, Goto Y, Vincent R, van Eeden SF. 2002. Interaction of alveolar macrophages and airway epithelial cells following exposure to particulate matter produces mediators that stimulate the bone marrow. Am J Respir Cell Mol Biol 27:34–41.
   PubMed Web of Science ®Google Scholar
• Goto Y, Hogg JC, Shih CH, Ishii H, Vincent R, van Eeden SF. 2004. Exposure to ambient particles accelerates monocyte release from the bone marrow in atherosclerotic rabbits. Am J Physiol Lung Cell Mol Physiol 287:L79–L85.
   PubMed Web of Science ®Google Scholar
• Huang C, Li J, Costa M, Castranova V, Vallyathan V, Ju G, Shi X. 2001. EInvolvement of hydrogen peroxide in nickel subsulfide induced NFAT activation. Cancer Res 61:8051–8057.
   PubMed Web of Science ®Google Scholar
• Lall R, Thurston GD. 2006. Identifying and quantifying transported vs. local sources of New York City PM2.5 fine particulate matter air pollution. Atmos Environ 40(Suppl 2):S333–S346.
   Google Scholar
• Levis JE, Mossman BT. 2007. Cell-signaling pathways elicited by particulates. In: Donaldson K, Borm P, editors. Particle Toxicology. Boca Raton, FL: CRC Press Inc., 197–209.
   Google Scholar
• Lippmann M. 2010. Targeting the components most responsible for airborne particulate matter health risks. J Expo Sci Environ Epidemiol 20:117–118.
   PubMed Web of Science ®Google Scholar
• Lippmann M, Chen LC. 2009. Health effects of concentrated ambient air particulate matter (CAPs) and its components. Crit Rev Toxicol 39:865–913.
   PubMed Web of Science ®Google Scholar
• Lippmann M, Gordon T, Chen LC. 2005. Effects of subchronic exposures to concentrated ambient particles (CAPs) in mice. I. Introduction, objectives, and experimental plan. Inhal Toxicol 17:177–187.
   PubMed Web of Science ®Google Scholar
• Lippmann M, Ito K, Hwang JS, Maciejczyk P, Chen LC. 2006. Cardiovascular effects of nickel in ambient air. Environ Health Perspect 114:1662–1669.
   PubMed Web of Science ®Google Scholar
• Maciejczyk P, Chen LC. 2005. Effects of subchronic exposures to concentrated ambient particles (CAPs) in mice. VIII. Source-related daily variations in in vitro responses to CAPs. Inhal Toxicol 17:243–253.
   PubMed Web of Science ®Google Scholar
• Maciejczyk P, Zhong M, Li Q, Xiong S, Nadziejko C, Chen LC. 2005. The design of a concentrated ambient particulate matter exposure system for biometric telemetry monitoring. Inhal Toxicol 17:189–197.
   PubMed Web of Science ®Google Scholar
• Mauderly JL, Chow JC. 2008. Health effects of organic aerosols. Inhal Toxicol 20:257–288.
   PubMed Web of Science ®Google Scholar
• NOAA. 2010. “Air Resources Laboratory—HYSPLIT—Hybrid Single-Particle Lagrangian Integrated Trajectory model.” http://www.arl.noaa.gov/HYSPLIT_info.php (Accessed June 17, 2010).
   Google Scholar
• Peltier RE, Lippmann M. 2010. Residual oil combustion: 2. Distributions of airborne nickel and vanadium within New York City. J Expo Sci Environ Epidemiol 20:342–350.
   PubMed Web of Science ®Google Scholar
• Peltier RE, Hsu SI, Lall R, Lippmann M. 2009. Residual oil combustion: a major source of airborne nickel in New York City. J Expo Sci Environ Epidemiol 19:603–612.
   PubMed Web of Science ®Google Scholar
• Porter DW, Leonard SS, Castranova V. 2007. Particles and cellular oxidative and nitrosative stress. In: Donaldson K, Borm P, editors. Particle Toxicology. Boca Raton, FL: CRC Press Inc., 119–138.
   Google Scholar
• Qin Y, Kim E, Hopke PK. 2006. The concentrations and sources of PM2.5 in Metropolitan New York City. Atmos Environ 40:S312–S332.
   Web of Science ®Google Scholar
• Schlesinger RB. 2007. The health impact of common inorganic components of fine particulate matter (PM2.5) in ambient air: A critical review. Inhal Toxicol 19:811–832.
   PubMed Web of Science ®Google Scholar
• Thurston GD, Spangler SD. 1985. A quantitative assessment of source contributions to inhalable particle matter pollution in metropolitan Boston. Atmos. Environ 19:9–25.
   Web of Science ®Google Scholar
• US Environmental Protection Agency. 2004. Air quality criteria for particulate matter. Research Triangle Park, NC: National Center for Environmental Assessment–RTP Office. Report No. EPA/600/P-99/002aF. October 2004.
   Google Scholar
• Zhang Z, Chau PY, Lai HK, Wong CM. 2009. A review of effects of particulate matter-associated nickel and vanadium species on cardiovascular and respiratory systems. Int J Environ Health Res 19:175–185.
   PubMed Web of Science ®Google Scholar