References
- Mattson M P. Cellular actions of b˜-amyloid precursor protein and its soluble and fibrillogenic derivatives. Physiol Rev 1997; 77: 1081–1132
- Arispe N, Rojas E, Pollard H B. Alzheimer disease amyloid p protein forms calcium channals in bilayer membranes: Blockade by tromethamine and aluminum. Proc Natl Acad Sci USA. 1993; 90: 567–571
- Ye C, Ho-Pao C L, Kanazirska M, Quinn S, Rogers K, Seidman C E, Seidman J G, Brown E M, Vassilev P M. Amyloid-b˜ proteins activate Ca2+-permeable channels through calcium-sensing receptors. J Neurosci Res 1997; 47: 547–554
- Ueda K, Shinohara S, Yagami T, Asakura K, Kawasaki K. Amyloid p protein potentiates Ca2+ influx through L-type voltage-sensitive Ca2+ channels: a possible involvement of free radicals. J Neurochem 1997; 68: 265–271
- Zhou Y, Gopalakrishnan V, Richardson J S. Actions of neurotoxic b˜-amyloid on calcium homeostasis and viability of PC12 cells are blocked by antioxidants but not by calcium channel antagonists. J Neurochem 1996; 67: 1419–1425
- Wallace M A. Effects of Alzheimer's disease-related b˜ amyloid protein fragments on enzymes metabolizing phosphoinositides in brain. Biochim Biophys Acta 1994; 1227: 183–187
- Singh I N, Sorrentino G, Kanfer J N. Amyloid b˜ protein (25–35) stimulation of phospholipase C in LA-N-2 cells. J Neurochem 1997; 69: 252–258
- Ishikawa H, Ozawa H, Saito T, Takahata N, Takemura H. Calcium mobilization evoked by amyloid b˜-protein involves inositol 1, 4, 5-trisphosphate production in human platelets. Life Sci 1998; 62: 705–713
- Fowler C J, Brännström G, Ahlgren P C, Florvall L, Åkerman K EO. Inhibition of inositol 1, 4, 5-trisphosphate-5-phosphatase by micromolar concentrations of disulfiram and its analogues. Biochem J 1993; 289: 853–859
- Ida N, Masters C L, Beyreuther K. Rapid cellular uptake of Alzheimer amyloid b˜A4 peptide by cultured human neuroblastoma cells. FEBS Letts 1996; 394: 174–178
- Hammad S M, Ranganathan S, Loukinova E, Twal W O, Argraves W S. Interaction of apolipoprotein b˜-amy-loid b˜-peptide complex with low density lipoprotein receptor-related protein-2/megalin. A mechanism to prevent pathological accumulation of amyloid b˜-peptide. J Biol Chem 1997; 272: 18644–18649
- Fowler C J, Ando Y, Tiger G. Comparison of the effects of hydrogen peroxide, 4-hydroxy-2-nonenal and b˜-amyloid(25–35) upon calcium signalling. Neurochem Intemat 1998; 33: 161–172
- Shen C L, Murphy R M. Solvent effects on self-assembly of beta-amyloid peptide. Biophys J 1995; 69: 640–651
- Fowler C J, Brännström G. Kinetic and inhibitor profiles of soluble and particulate inositol-(1, 4, 5)-trisphosphate-5-phosphatase from GH3 and IMR-32 cells. Biochem J 1990; 271: 735–742
- Markwell M AK, Haas S M, Bieber L L, Tolbert N E. A modification of the Lowry procedure to simplify protein determination in membrane and lipoprotein samples. Analyt Biochem 1978; 87: 206–210
- Hensley K, Carney J M, Mattson M P, Aksenova M, Harris M, Wu J F, Floyd R, Butterfield D A. A model for b˜-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease. Proc Natl Acad Sci USA 1994; 91: 3270–3274
- Turner R S, Suzuki N, Chyung A S, Younkin S G, Lee V M. Amyloids b˜40 and b˜42 are generated intracellularly in cultured human neurons and their secretion increases with maturation. J Biol Chem 1996; 111: 8966–8970
- Rosenblum W I. The presence, origin, and significance of Ap peptide in the cell bodies of neurons. J Neuropathol Exp Neurol 1999; 58: 575–581
- Wilson C A, Doms R W, Lee V M-Y. Intracellular APP processing and AP production in Alzheimer disease. J Neuropathol Exp Neurol 1999; 58: 787–794
- Canevari L, Clark J B, Bates T E. b˜-Amyloid fragment 25–35 selectively decreases complex IV activity in isolated mitochondria. FEBS Letts 1999; 457: 131–134
- Guse A H, Greiner E, Emmrich F, Brand K. Mass changes of inositol 1, 3, 4, 5, 6-pentakisphosphate and inositol hexakisphosphate during cell cycle progression in rat thymocytes. J Biol Chem 1993; 268: 7129–33
- Stuart J A, Anderson K L, French P J, Kirk C J, Michell R H. The intracellular distribution of inositol polyphosphates in HL60 promyeloid cells. Biochem J 1994; 303: 517–525
- Larsson O, Barker C J, Sjöholm Å, Carlqvist H, Michell R H, Bertorello A, Nilsson T, Honkanen R E, Mayr G W, Zwiller J, Berggren P-O. Inhibition of phosphatases and increased Ca2+ channel activity by inositol hexakisphosphate. Science 1997; 278: 471–474