References
- Brint EK, Xu D, Liu H, et al. (2004). ST2 is an inhibitor of interleukin 1 receptor and Toll-like receptor 4 signaling and maintains endotoxin tolerance. Nat Immunol 5:373–9
- Brunner M, Krenn C, Roth G, et al. (2004). Increased levels of soluble ST2 protein and IgG1 production in patients with sepsis and trauma. Intensive Care Med 30:1468–73
- Dieplinger B, Januzzi JrJL, Steinmair M, et al. (2009). Analytical and clinical evaluation of a novel high-sensitivity assay for measurement of soluble ST2 in human plasma – the Presage ST2 assay. Clin Chim Acta 409:33–40
- Januzzi JrJL, Peacock WF, Maisel AS, et al. (2007). Measurement of the interleukin family member ST2 in patients with acute dyspnea: results from the PRIDE (Pro-Brain Natriuretic Peptide Investigation of Dyspnea in the Emergency Department). study. J Am Coll Cardiol 50:607–13
- Kobashigawa J, Crespo-Leiro MG, Ensminger SM, et al. (2011). Report from a consensus conference on antibody-mediated rejection in heart transplantation. J Heart Lung Transplant 30:252–69
- Liu J, Buckley JM, Redmond HP, Wang JH. (2010). ST2 negatively regulates TLR2 signaling, but is not required for bacterial lipoprotein-induced tolerance. J Immunol 184:5802–8
- Pascual-Figal DA, Garrido IP, Blanco R, et al. (2011). Soluble ST2 is a marker for acute cardiac allograft rejection. Ann Thorac Surg 92:2118–24
- Pietra BA, Wiseman A, Bolwerk A, et al. (2000). CD4 T cell-mediated cardiac allograft rejection requires donor but not host MHC class II. J Clin Invest 106:1003–10
- Rehman SU, Mueller T, JanuzziJr JL. (2008). Characteristics of the novel interleukin family biomarker ST2 in patients with acute heart failure. J Am Coll Cardiol 52:1458–65
- Sabatine MS, Morrow DA, Higgins LJ, et al. (2008). Complementary roles for biomarkers of biomechanical strain ST2 and N-terminal prohormone B-type natriuretic peptide in patients with ST-elevation myocardial infarction. Circulation 117:1936–44
- Sanada S, Hakuno D, Higgins LJ, et al. (2007). IL-33 and ST2 comprise a critical biomechanically induced and cardioprotective signaling system. J Clin Invest 117:1538–49
- Schmitz J, Owyang A, Oldham E, et al. (2005). IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. Immunity 23:479–90
- Seki K, Sanada S, Kudinova AY, et al. (2009). Interleukin-33 prevents apoptosis and improves survival after experimental myocardial infarction through ST2 signaling. Circ Heart Fail 2:684–91
- Shah RV, Chen-Tournoux AA, Picard MH, et al. (2009). Serum levels of the interleukin-1 receptor family member ST2, cardiac structure and function, and long-term mortality in patients with acute dyspnea. Circ Heart Fail 2:311–19
- Stewart S, Winters GL, Fishbein MC, et al. (2005). Revision of the 1990 working formulation for the standardization of nomenclature in the diagnosis of heart rejection. J Heart Lung Transplant 24:1710–20
- Tay SS, Plain KM, Bishop GA. (2009). Role of IL-4 and Th2 responses in allograft rejection and tolerance. Curr Opin Organ Transplant 14:16–22
- Turnquist HR, Sumpter TL, Tsung A, et al. (2008). IL-1beta-driven ST2L expression promotes maturation resistance in rapamycin-conditioned dendritic cells. J Immunol 181:62–72
- Waaga AM, Gasser M, Kist-Van Holthe JE, et al. (2001). Regulatory functions of self-restricted MHC class II allopeptide-specific Th2 clones in vivo. J Clin Invest 107:909–16
- Wang J, Guo Z, Dong Y, et al. (2003). Role of 4-1BB in allograft rejection mediated by CD8+ T cells. Am J Transplant 3:543–51
- Weir RA, Miller AM, Murphy GE, et al. (2010). Serum soluble ST2: a potential novel mediator in left ventricular and infarct remodeling after acute myocardial infarction. J Am Coll Cardiol 55:243–50
- Yin H, Li XY, Jin XB, et al. (2010). IL-33 prolongs murine cardiac allograft survival through induction of TH2-type immune deviation. Transplantation 89:1189–97