References
- Wiman B, Hamsten A. Decreased fibrinolytic activity in thrombotic disease: An update. Hypercoagulable States: Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 152–154
- Seghatchian MJ, Baglin T, Samama MM. Hematological disorders and stroke. Hypercoagulable States. Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 277–283
- Hillarp A, Zoller B, Dahlback B. Activated protein C resistance as a basis for venous thrombosis. Am J Med 1996; 101: 534–540
- Salzman E, Hirsh J. The epidemiology pathogenesis and natural history of venous thrombosis. Hemostasis and Thrombosis Basic Principles and Clinical Practice3rd ed., RW Colman, J Hirsh, VJ Marder, EW Salzman. Lippincott-Raven, Hagerstown, NJ 1994; 1275–1296
- Gough SCL, Prentice CRM. Hypercoagulability and diabetes mellitus. Hypercoagulable States: Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 267–276
- Plesner T, Bergen H, Hansen NE. Paroxysmal nocturnal hemoglobinuria. Pathogenesis and clinical manifestations of thrombosis. Hypercoagulable States: Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 285–292
- Verdy E, Girot R. Sickle cell disease. Hypercoagulable States: Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 409–414
- Eldor A, Maclouf J, Goldfarb A, Durst R, Guillin MC, Rachmilewitz EA. A life-long hypercoagulable state in beta thalasemia major patient. Hypercoagulable States: Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 415–421
- Green D, Chen D. Spinal cord injury: Pathophysiology of hypercoagulability and clinical management. Hypercoagulable States: Fundamental Aspects Acquired Disorders and Congenital Thrombophilia, MJ Seghatchian, MM Samama, SP Hecker. CRC Press, Boca Raton 1996; 293–302
- Kitzmiller JL, Benirschke K. Immunofluorescent study of placental bed vessels in preeclampsia. Am J Obstet Gynecol 1973; 115: 248
- Fujikura T, Benson RC. Placentitis and fibrous occlusion of fetal vessels in the placentas of stillborn infants. Am J Obstet Gynecol 1964; 89: 225–232
- Naeye RL. Placental infarction leading to fetal or neonatal death. Obstet Gynecol 1977; 50: 583–588
- Gruenwald P. Abnormalities of placental vascularity in relation to intrauterine deprivation and retardation of fetal growth. New York State J Med 1961; 61: 1508–1514
- Fox H. The histopathology of placental insufficiency. J Clin Path 1976; 10: 1–8
- Arias F, Rodriguez L, Rayne SC, Kraus FT. Maternal placental vasculopathy and infection: Two distinct subgroups among patients with preterm labor and preterm ruptured membranes. Am J Obstet Gynecol 1993; 168: 585–591
- Arias F, Victoria A, Cho K, Kraus FT. Placental histology and clinical characteristics of patients with preterm premature rupture of membranes. Obstet Gynecol 1997; 89: 265–271
- Beaconsfield R, Birdwood G. Placenta—The largest human biopsy. Pergamon Press, New York 1982
- Kraus FT. Placental thrombi and related problems. Sem Diagn Pathol 1993; 10: 275–283
- Redline RW, Pappini A. Fetal thrombotic vasculopathy: The clinical significance of extensive avascular villi. Hum Pathol 1995; 26: 8085
- Benirschke K, Kaufmann P. Pathology of the Human Placenta3 ed. Springer Verlag, New York 1995; 245–250
- Kraus FT. Placenta: Thrombosis of fetal stem vessels with fetal thrombotic vasculopathy and chronic villitis. Pediat Pathol Lab Med 1996; 16: 143–148
- Dizon-Townson DS, Meline L, Nelson LM, Varner M, Ward K. Fetal carriers of the factor V Leiden mutation are prone to miscarriage and placental infarction. Am J Obstet Gynecol 1997; 177: 402405
- Rai RS, Regan L, Chitolie A, Donald JG, Cohen H. Placental thrombosis and second trimester miscarriage in association with activated protein C resistance. Br J Obstet Gynaecol 1996; 103: 842–844
- Rotmensch S, Libetati M, Mittelmann M, Ben-Rafael Z. Activated protein C resistance and adverse pregnancy outcome. Am J Obstet Gynecol 1997; 177(1)170–173
- Dekker GA, de Vries JIP, Doelitzsch PM, Huijgens PC, von Blomberg BME, Jakobs C, Geijn HP. Underlying disorders associated with severe early-onset preeclampsia. Am J Obstet Gynecol 1995; 173: 1042–1048
- Lindoff C, Ingemarsson I, Martinsson G, Segelmark M, Thysell H, Astedt B. Preeclampsia is associated with a reduced response to activated protein C. Am J Obstet Gynecol 1997; 176: 457–460
- Espana F, Gilabert J, Aznar J, Estelles A, Kobayashi T, Griffin JH. Complexes of activated protein C with alpha1-antitrypain in normal pregnancy and in severe preeclampsia. Am J Obstet Gynecol 1991; 164: 1310–1316
- Halligan A, Bonnar J, Sheppard B, Darling M, Walshe J. Haemostatic, fibrinolytic and endothelial variables in normal pregnancies and pre-eclampsia. Br J Obstet Gynaecol 1994; 101: 488–492
- Romero R, Lockwood C, Oyarzun E, Hobbins JC. Toxemia: New concepts in an old disease. Seminars Perinatol 1988; 12: 302–323
- Weiner CP. Preeclampsia-eclampsia syndrome and coagulation. Hyperten Preg 1991; 18: 713–726
- de Vries JIP, Dekker GA, Huijgens PC, Jakobs C, Blomberg BME, Van Geijn HP. Hyperhomocysteinaemia and protein S deficiency in complicated pregnancies. Br J Obstet Gynecol. 1997; 104: 1248–1254
- Thorarensen O, Ryan S, Hunter J, Younkin DP. Factor V mutation: An unrecognized cause of hemiplegic cerebral palsy neonatal stroke and placental thrombosis. Ann Neurol 1997; 42: 372–375
- Kraus FT. Cerebral palsy and thrombi in placental vessels of the fetus. Insights from litigation. Hum Pathol 1997; 28: 246–248
- Rees D, Cox M, Clegg J. World Distribution of Factor V Leiden. Lancet 1995; 346: 1133–1134
- Guyen N, Hezard D, Comillet P, Potron G. ROC curves to evaluate the performance of “clotting” methods for Factor V Leiden. Br Haematolog 1997; 96: 872
- Poort SR, Rosendaal FR, Reitsma PH, Bertina RM. A common genetic variation in the 3′-untranslated region of the prothrombin gene is associated with elevated plasma prothrombin levels and an increase in venous thrombosis. Blood 1996; 88: 3698–3703
- Rajkovic A, Catalano PM, Malinow MR. Elevated homocyt(e)ine levels with preeclampsia. Obstet Gynecol 1997; 90: 168–171
- Dizon-Townson D, Meline L, Nelson LM, Varner M, Ward K. Fetal carriers of the Factor V Leiden mutation are prone to miscarriage and placental infarct. Am J Obstet Gynecol 1997; 176: 883–886
- Dahlback B. Inherited thrombophilia: Resistance to activated protein C as a pathogenic factor of venous thromboembolism. Blood 1995; 85: 607–614