REFERENCES
- Backer H, Lindsey RJ. The laboratory rat – research application, NRC (Vol. I, II). New York: Academic Press, 1980.
- Barja G. Rate of generation of oxidative stress-related damage and animal longevity. Free Radic Biol Med. 2002;23:1167–1172.
- Bathum L, Petersen I, Christiansen L, Konieczna A, Sørensen TI, Kyvik KO. Genetic and environmental influences on plasma homocysteine: results from a Danish twin study. Clin Chem. 2007;53:971–979.
- Bridges AB, Scott NA, Pary GJ, Belch JJ. Age, sex, cigarette smoking indices of radical activity in healthy humans. Eur J Med. 1993;2:205–208.
- Camougrand N, Rigoulet M. Aging and oxidative stress: studies of some genes involved both in aging and in response to oxidative stress. Resp Physiol. 2001;128:393–401.
- Eikelboom JW, Lonn E, Genest JJ, Hankey G, Yusuf S. Homocysteine and cardiovascular disease: a critical review of the epidemiologic evidence. Ann Intern Med. 1999;131:363–375.
- Glaz J, Balakrishnan N. (Eds.). Scan statistics and applications, p. 368. Book review. New York: Springer, 1999.
- Harman D. Aging: a theory based on free radical and radiation chemistry. J Gerontol. 1956;11:298–300.
- Heizer M, Rosendaal FR, Bloom H, Gerrits WBJ, Bos GMJ. Hyperhomocysteinemia and venous thrombosis: a meta-analysis. Thromb Haemost. 1998;80:874–877.
- Kullo IJ, Ding K, Boerwinkle E, Turner ST, Mosley Jr TH, Kardia SL, de Andrade M. Novel genomic loci influencing plasma homocysteine levels. Stroke. 2006;37:1703–1709.
- Lipton SA, Kim WK, Choi YB, Kumar S, D’Emilla DM, Rayudu PV, Arnelles DR, Stamler JS. Neurotoxicity associated with dual actions of homocysteine at the N-methyl-D-aspartate receptor. Proc Natl Acad Sci. 1997;94:5923–5928.
- Lovell MA, Xie C, Markesbery WR. Acrolein, a product of lipid peroxidation, inhibits glucose and glutamate uptake in primary neuronal cultures. Free Radic Biol Med. 2000;29(8):714–720.
- Mattevi A. To be or not to be an oxidase: challenging the oxygen reactivity of flavoenzymes. Trends Biochem Sci. 2006;31:276–283.
- Nakiba T, Cui Z, Masuda J. The stroke prone spontaneously hypertensive rat: how good is it as a model for cerebrovascular diseases. Cell Mol Neurobiol. 2004;24(5):639–644.
- Perez SC, Vianna LM. Favorable effect of pyridoxin and folic acid supplementation of stroke-prone rats. Archivos de Neurociencias. 2005;03(10):146–149.
- Scherle W. A simple method for volumetry of organs in quantitative stereology. Mikroskopie. 1970;26:57–60.
- Sohal RS, Sohal BH, Orr WC. Mitochondril superoxide and hydrogen peroxide generation, protein oxidative damage and longevity in different species of files. Free Radic Biol Med. 1995;199:199–504.
- Tascilar N, Ekem S, Aciman E, Ankarali H, Mungan G, Ozen B, Unal A. Hyperhomocysteinemia as an independent risk factor for cardioembolic stroke in the Turkish population. Tohoku J Exp Med. 2009;218(4):293–300.
- Ullegaddi R, Powers HJ, Gariballa SE. B-group vitamin supplementation mitigates oxidative damage after acute ischaemic stroke. Clin Sci. 2004;107(5):477–484.
- Ullegaddi R, Powers JH, Gariballa SE. Antioxidant supplementation with or without B-group vitamins after acute ischemic stroke: a randomized controlled trial. J Parenter Enteral Nutr. 2006;30(2):108–114.
- Vianna LM, Paiva ACM, Paiva TB. Treatment with vitamin D lowers blood pressure of spontaneously hypertensive rats. Genetic Hypertens. 1992;218:589–591.
- Vianna LM, Teixeira F, Mizurinni D. Pyridoxine and folic acid combined therapy provides the best physiologic response. Ann Nutr Metab. 2003;47:563–564.
- Whishaw IQ, Kolb B. (Eds.). The behavior of the laboratory rat – a handbook with tests, p. 504. New York: Oxford: University Press, 2005.
- Wickens AP. Ageing and the free radical theory. Respir Physiol. 2001;128:379–391.