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Review Article

Screening tests for inflammatory activity: applications in rheumatology

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Pages 469-477 | Received 10 Mar 2009, Accepted 07 Jul 2009, Published online: 02 Jan 2014

References

  • Volanakis JE. Acute-phase proteins in rheumatic disease. In: Koopmann WJ, Moreland LW, editors. Arthritis and allied con-ditions. 15th ed. Philadelphia: Lippincott William & Wilkins; 2005. p. 504–16.
  • Kushner I, Gabay C. Acute-phase proteins and other systemic responses to inflammation. N Engl J Med. 1999;340:448–54.
  • James K. Cellular and humoral mediators of inflammation: non-specific humoral response to inflammation. In: McClatchey KD, editor. Clinical laboratory medicine. 2nd ed. Philadelphia: Lippincott Willians & Wilkins; 2002. p. 1430–46.
  • de Carvalho JF, Hanaoka B, Szyper-Kravitz M, Shoenfeld Y. C-reactive protein and its implications in systemic lupus erythe-matosus. Acta Reumatol Port. 2007;32:317–22.
  • Gershov D, Kim S, Brot N, Elkon KB. C-reactive protein binds to apoptotic cells, protects the cells from assembly of the terminal complement components, and sustains an anti-inflammatory innate immune response: implications for systemic autoimmu-nity. J Exp Med. 2000;192(9):1353–63.
  • Mold C, Baca R, Du Clos TW. Serum amyloid P component and C-reactive protein opsonize apoptotic cells for phagocytosis through Fc. receptors. J Autoimmun. 2002;19:147–54.
  • Atkinson JP. C-reactive protein: a rheumatologist's friend revis-ited. Arthritis Rheum. 2001;44(5):995–6.
  • Sheldon J. Laboratory testing in autoimmune rheumatic diseases. Best Pract Res Clin Rheumatol. 2004;18(3):249–69.
  • Kushner I. C-reactive protein elevation can be caused by condi-tions other than inflammation and may reflect aging. Cleve Clin J Med. 2001;68:535.
  • Kavanaugh A. The role of laboratory in the evaluation of rheu-matic diseases. Clin Cornerstone. 1999;2(2):11–25.
  • Rifai N, Ridker PM. High-sensitivity C-reactive protein: a novel and promising marker of coronary heart disease. Clin Chem. 2001;47(3):403–11.
  • Salmon JE, Roman MJ. Subclinical atherosclerosis in rheumatoid arthritis and systemic lupus erythematosus. Am J Med. 2008; 121(10A):53–8.
  • Collares GB, Vidigal PG. Recomendagoes para o uso da veloc-idade de hemossedimentação. Rev Med Minas Gerais. 2004; 14(1):52–7.
  • Uhlar CM, Whitehead AS. Serum amyloid A, the major vertebrate acute-phase reactant. Eur J Biochem. 1999;265:501–23.
  • Jense LE, Whitehead AS. Regulation of serum amyloid A protein expression during acute-phase response. Biochem J. 1998; 334:489–503.
  • Marhaug G, Dowton SB. Serum Amyloid A: an acute phase apolipoprotein and precursor of AA amyloid. Baillieres Clin Rheumatol. 1994;8(3):553–73.
  • Hochepied T, Berger FG, Baumann H, Libert C. al-acid glyco-protein: an acute-phase protein with inflammatory and immuno-modulating properties. Cytokine Growth Factor Rev. 2003; 14:25–34.
  • Fournier T, Medjoubi-N N, Pourquet D. Alpha-l-acid glycopro-tein. Biochim Biophys Acta. 2000;1482:157–171.
  • O'Connel TX, Horite TJ, Kasravi B. Understanding and inter-preting serum protein electrophoresis. Am Fam Phys. 2005;71(1): 105–12.
  • Meijvis SC, Endeman H, Geers AB, ter Borg RI. Extremely high serum ferritin levels as diagnostic tool in adult-onset Still's dis-ease. Neth J Med. 2007;65(6):212–4.
  • Yamanishi H, Kimura S, Hata N, Iyama S, Kanakura Y, Iwatani Y. Evaluation of a model of latent pathologic factor in relation to serum ferritin elevation. Clin Biochem. 2007;40: 359–64.
  • Schwarz-Eywill M, Heilig B, Bauer H, Breitbart A, Pezzutto A. Evaluation of serum ferritin as a marker for adult Still's disease activity. Ann Rheum Dis. 1992;51:683–5.
  • Lee MH, Means RT Jr. Extremely elevated serum ferritin levels in a university hospital: associated diseases and clinical signifi-cance. Am J Med. 1995;98:566–71.
  • Koorts AM, Viljoen M. Fenitin and ferritin isoforms I: structure-function relationships, synthesis, degradation and secretion. Arch Physiol Biochem. 2007;113(1):30–54.
  • Koorts AM, Viljoen M. Fenitin and ferritin isoforms II: protec-tion against uncontrolled cellular proliferation, oxidative damage and inflammatory processes. Arch Physiol Biochem. 2007; 113(1):55–64.
  • Emery P, Gabay C, Kraan M, Gomez-Reino J. Evidence-based review of biologic markers as indicators of disease progression and remission in rheumatoid arthritis. Rheumatol Int. 2007; 27:793–806.
  • Matsui T, Kuga Y, Kaneko A, et al. Disease Activity Score 28 (DA528) using C-reactive protein underestimates disease activity and overestimates EULAR response criteria compared with DA528 using erythrocyte sedimentation rate in a large observa-tional cohort of rheumatoid arthritis patients in Japan. Ann Rheum Dis. 2007;66:1221–6.
  • Paulus HE, Brahn E. Is erythrocyte sedimentation rate the pref-erable measure of the acute phase response in rheumatoid arthritis? [editorial]. J Rheumatol. 2004;31(5):838–40.
  • Shovman 0, Gilburd B, Zandman-Goddard G, et al. The diag-nostic utility of anti-cyclic citrullinated peptide antibodies, matrix metalloproteinases-3, rheumatoid factor, erythrocyte sedimenta-tion rate, and C-reactive protein in patients with erosive and non-erosive rheumatoid arthritis. Clin Dev Immunol. 2005;12(3): 197–202.
  • Young-Min S, Cawston T, Marshall N, et al. Biomarkers predict radiographic progression in early rheumatoid arthritis and per-form well compared with traditional markers. Arthritis Rheum. 2007 ;56(10) : 3236–47.
  • Ates A, Tfirkopar N, Olmez U, et al. Serum pro-matrix metalloproteinase-3 as an indicator of disease activity and severity in rheumatoid arthritis: comparison with traditional markers. Rheumatol Int. 2007;27:715–22.
  • Hameed B, Pilcher J, Heron C, Kiely PD. The relation between composite ultrasound measures and the DA528 score, its com-ponents and acute phase markers in adult RA. Rheumatology. 2008;47:476–80.
  • Badolato R, Oppenheim JJ. Role of cytokines, acute-phase pro-teins, and chemokines in the progression of rheumatoid arthritis. Semin Arthritis Rheum. 1996;26(2):526–38.
  • Cutolo M, Montecucco CM, Cavagna L, Caporali R, Capellino S, Montagna P, et al. Serum cytokines and steroidal hormones in polymyalgia rheumatica and elderly-onset rheumatoid arthritis. Ann Rheum Dis. 2006;65(11):1438–43.
  • Karlson EW, Chibnik LB, Tworoger SS, Lee IM, Buring JE, Shadick NA. Biomarkers of inflammation and development of rheumatoid arthritis in women from two prospective cohort studies. Arthritis Rheum. 2009;60(3):641–52.
  • Vignes S, Le Moël G, Fautrel B, Wechsler B, Godeau P, Piette JC. Percentage of glycosilated serum ferritin remains low throughout the course of adult-onset Still's disease. Ann Rheum Dis. 2000;59:347–50.
  • Hamidou MA, Denis M, Barbarot S, Boutoille D, Belizna C, Le Moël G. Usefulness of glycosilated ferritin in atypical pre-sentations of adult-onset Still's disease (letter). Ann Rheum Dis. 2004;63:605.
  • Jung SY, Park MC, Park YB, Lee SK. Serum amyloid A as a useful indicator of disease activity in patients with ankylosing spondylitis. Yonsei Med J. 2007;48(2):218–24.
  • Ozgocmen S, Godekmerdan A, Ozkurt-Zengin F. Acute-phase response, clinical measures and disease activity in ankylosing spondylitis. Joint Bone Spine. 2007;74:249–53.
  • Kaya T, Bal S, Gunaydin R. Relationship between the severity of enthesitis and clinical and laboratory parameters in patients with ankylosing spondylitis. Rheumatol Int. 2007;27:323–7.
  • Yildirim K, Erdal A, Karatay S, Melikoglu MA, Ugur M, Senel K. Relationship between some acute phase reactants and the bath ankylosing spondylitis disease activity index in patients with ankylosing spondylitis. South Med J. 2004;97(4):350–3.
  • Lange U, Boss B, Teichmann J, Klör HU, Neeck G. Serum Amyloid A-an indicator of inflammation in ankylosing spon-dylitis. Rheumatol Int. 2000;19:119122.
  • Visvanathan S, Wagner C, Marini JC, et al. Inflammatory bio-markers, disease activity and spinal disease measures in patients with ankylosing spondylitis after treatment with infliximab. Ann Rheum Dis. 2008;67:511–7.
  • Romero-Sánchez C, Robinson WH, Tomooka BH, et al. Identi-fication of acute phase reactants and cytokines useful for moni-toring infliximab therapy in ankylosing spondylitis. Clin Rheumatol. 2008;27:1429–35.
  • Roodpeyma S, Karnali Z, Zare R. Rheumatic fever: the rela-tionship between clinical manifestations and laboratory tests. J Pediatr Child Health. 2005;41:97–100.
  • Ellis ME, Ralston S. The ESR in the diagnosis and management of the polymyalgia rheumatica/giant-cell arteritis syndrome. Ann Rheum Dis. 1983;42:168–70.
  • Helfgot S, Kieval RI. Polymyalgia rheumatica in patients with a normal erythrocyte sedimentation rate. Arthritis Rheum. 1996; 39:304–7.
  • Caliani L, Paira SO. Polymyalgia rheumatica in patients with a normal erythrocyte sedimentation rate: comment on the article by Helgot and Kieval (letter). Arthritis Rheum. 1997;40(9):1725–6.
  • Cimmino MA, Accardo S, Scudeletti M, Indiveri F. The diag-nosis of polymyalgia rheumatica in patients with a low erythro-cyte sedimentation rate: comment on the article by Helfgot and Kieval (letter). Arthritis Rheum. 1997;40(9):1726–7.
  • Gaitonde S, Samols D, Kushner I. C-reactive protein and systemic lupus erythematosus. Arthritis Rheum. 2009;59(12): 1814–20.
  • Gitlin JD, Colten HR. Molecular biology of the acute-phase plasma proteins. In: Pick E, Landy M, editors. Lymphokines, vol. 14. San Diego: Academic Press; 1987. p. 123–53.

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