TERT distal promoter GC islands are critical for telomerase and together with DNMT3B silencing may serve as a senescence-inducing agent in gliomas

Naz Şerifoğlua National Nanotechnology Research Center (UNAM), Bilkent University, Ankara, Turkey;b Interdisciplinary Graduate Program in Neuroscience, Aysel Sabuncu Brain Research Center, Bilkent University, Ankara, Turkey;c Institute for Research on Cancer and Aging of Nice, French National Centre for Scientific Research, Paris, FranceView further author information

Begün Erbabaa National Nanotechnology Research Center (UNAM), Bilkent University, Ankara, TurkeyView further author information

Michelle M. Adamsa National Nanotechnology Research Center (UNAM), Bilkent University, Ankara, Turkey;b Interdisciplinary Graduate Program in Neuroscience, Aysel Sabuncu Brain Research Center, Bilkent University, Ankara, Turkey;d Department of Psychology, Bilkent University, Ankara, TurkeyView further author information

Ayça Arslan-Ergüla National Nanotechnology Research Center (UNAM), Bilkent University, Ankara, TurkeyCorrespondence[email protected]
View further author information

TERT distal promoter GC islands are critical for telomerase and together with DNMT3B silencing may serve as a senescence-inducing agent in gliomas

supplementary_file_july22.docx

Information for

Open access

Opportunities

Help and information

Your download is now in progress and you may close this window

Login or register to access this feature

TERT distal promoter GC islands are critical for telomerase and together with DNMT3B silencing may serve as a senescence-inducing agent in gliomas

supplementary_file_july22.docx

Reprints and Corporate Permissions

Academic Permissions

Related research

To cite this article:

Download citation

Information for

Open access

Opportunities

Help and information

Keep up to date