Iguratimod prevents renal fibrosis in unilateral ureteral obstruction model mice by suppressing M2 macrophage infiltration and macrophage–myofibroblast transition

Yueyuan Zhoua Department of General Medicine, Xiangya Hospital, Central South University, Changsha, China;b National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, China

https://orcid.org/0000-0002-3765-2585 View further author information

Zhilan Lia Department of General Medicine, Xiangya Hospital, Central South University, Changsha, China;b National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, ChinaView further author information

Shenyi Yuc Department of Rheumatology and Immunology, Zhuzhou Hospital Affiliated to Xiangya Medical College, Central South University, Zhuzhou, ChinaView further author information

Xuan Wanga Department of General Medicine, Xiangya Hospital, Central South University, Changsha, China;b National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, ChinaView further author information

Tingting Xiea Department of General Medicine, Xiangya Hospital, Central South University, Changsha, China;b National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, ChinaView further author information

Weiru Zhanga Department of General Medicine, Xiangya Hospital, Central South University, Changsha, China;b National Clinical Research Center for Geriatric Disorders, Xiangya Hospital, Central South University, Changsha, ChinaCorrespondence[email protected]
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Iguratimod prevents renal fibrosis in unilateral ureteral obstruction model mice by suppressing M2 macrophage infiltration and macrophage–myofibroblast transition

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Iguratimod prevents renal fibrosis in unilateral ureteral obstruction model mice by suppressing M2 macrophage infiltration and macrophage–myofibroblast transition

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