Fusobacterium nucleatum promotes esophageal squamous cell carcinoma progression and chemoresistance by enhancing the secretion of chemotherapy-induced senescence-associated secretory phenotype via activation of DNA damage response pathway

Jian-Wei Zhanga Department of Gastroenterology, Renmin Hospital of Wuhan University, Wuhan, Hubei, PR China;b Key Laboratory of Hubei Province for Digestive System Diseases, Wuhan, Hubei, PR China;c Department of Gastroenterology, Chongqing University Cancer Hospital, Chongqing, PR ChinaView further author information

Dan Zhangd Department of Radiation and Medical Oncology, Zhongnan Hospital of Wuhan University, Wuhan, Hubei, PR ChinaView further author information

Hai-Sen Yina Department of Gastroenterology, Renmin Hospital of Wuhan University, Wuhan, Hubei, PR China;b Key Laboratory of Hubei Province for Digestive System Diseases, Wuhan, Hubei, PR ChinaView further author information

Han Zhanga Department of Gastroenterology, Renmin Hospital of Wuhan University, Wuhan, Hubei, PR China;b Key Laboratory of Hubei Province for Digestive System Diseases, Wuhan, Hubei, PR ChinaView further author information

Kun-Qiao Honga Department of Gastroenterology, Renmin Hospital of Wuhan University, Wuhan, Hubei, PR China;b Key Laboratory of Hubei Province for Digestive System Diseases, Wuhan, Hubei, PR ChinaView further author information

Jing-Ping Yuane Department of Pathology, Renmin Hospital of Wuhan University, Wuhan, Hubei, PR ChinaCorrespondence[email protected]
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Bao-Ping Yua Department of Gastroenterology, Renmin Hospital of Wuhan University, Wuhan, Hubei, PR China;b Key Laboratory of Hubei Province for Digestive System Diseases, Wuhan, Hubei, PR ChinaCorrespondence[email protected]
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Fusobacterium nucleatum promotes esophageal squamous cell carcinoma progression and chemoresistance by enhancing the secretion of chemotherapy-induced senescence-associated secretory phenotype via activation of DNA damage response pathway

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Fusobacterium nucleatum promotes esophageal squamous cell carcinoma progression and chemoresistance by enhancing the secretion of chemotherapy-induced senescence-associated secretory phenotype via activation of DNA damage response pathway

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