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Abstract
Extract
Coronary heart disease is classically considered to be a consequence of altered blood supply to the myocardium because of atherosclerosis. The CitationWHO (1958) definition of atherosclerosis is “a variable combination of changes in the intima of arteries consisting of the focal accumulation of lipids, complex carbohydrates, blood and blood products, fibrous tissue and calcium deposits, and associated with medial changes”. CitationFurman (1969) has stated that “atherogenesis is one problem, thrombogenesis a second, fatal arrhythmia perhaps a third, and that they may or may not be related. In the final analysis the conclusion seems inescapable that if atherogenesis could be prevented, thrombus formation would be of little or no significance in coronary artery disease, and myocardial infarction would virtually disappear”.