132
Views
0
CrossRef citations to date
0
Altmetric
Editorial

The Role of DNA Methylation in the Pathogenesis of Disease: What can Epigenome-Wide Association Studies Tell?

, &
Pages 5-7 | Received 25 Sep 2015, Accepted 05 Oct 2015, Published online: 23 Dec 2015
 

Financial & competing interests disclosure

The authors are supported by the Swedish Society for Medical Research (SSMF), the Kjell och Märta Beijers Foundation, the Göran Gustafssons Foundation and the Swedish Brain Foundation. The authors have no other relevant affiliations or financial involvement with any organization or entity with a financial interest in or financial conflict with the subject matter or materials discussed in the manuscript apart from those disclosed.

No writing assistance was utilized in the production of this manuscript.

Additional information

Funding

The authors are supported by the Swedish Society for Medical Research (SSMF), the Kjell och Märta Beijers Foundation, the Göran Gustafssons Foundation and the Swedish Brain Foundation. The authors have no other relevant affiliations or financial involvement with any organization or entity with a financial interest in or financial conflict with the subject matter or materials discussed in the manuscript apart from those disclosed. No writing assistance was utilized in the production of this manuscript.

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.