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Scandinavian Journal of Gastroenterology Volume 6, 1971 - Issue 5
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Original Article

Lactic Acid in Gastric Juice

D. W. Piper Dept. of Medicine, The University of Sydney and the Royal North Shore Hospital of Sydney, Sydney, N. S. W., Australia
,
A. D. Clarke Dept. of Medicine, The University of Sydney and the Royal North Shore Hospital of Sydney, Sydney, N. S. W., Australia
,
D. Whitecross Dept. of Medicine, The University of Sydney and the Royal North Shore Hospital of Sydney, Sydney, N. S. W., Australia
&
M. Croydon Dept. of Medicine, The University of Sydney and the Royal North Shore Hospital of Sydney, Sydney, N. S. W., Australia
Pages 465-469 | Received 10 Mar 1971, Accepted 05 Apr 1971, Published online: 25 Feb 2010
 
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Abstract

The conventional stimulants of gastric secretion (histamine, pentagastrin, and insulin) results in a fall in lactic acid concentration and usually a rise in lactic acid output. Agents that impose indirectly or directly a lactic acid load (glucose, fructose, xylitol, epinephrine) usually result in an increase in lactic acid concentration and output. It is considered that non-ionic diffusion plays little role in lactic acid secretion, and that the lactic acid content of gastric juice reflects the metabolic processes in the gastric mucosal cells.

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