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Occupational

Effect of Asthma Call-back Survey methodology changes on work-related asthma estimates, 19 states, 2007–2012

, MPH, ASPPH/CDC Public Health Fellow & , MD, MS, PhD
Pages 382-386 | Received 11 Jun 2015, Accepted 24 Sep 2015, Published online: 10 Feb 2016
 

Abstract

Objective: Asthma Call-back Survey methodology has been changed recently, as a new sampling design, weights calculation (2011–2012), and revised work-related asthma (WRA) section (2012) were implemented. To assess the effect of these changes on the WRA and possible WRA estimates among ever-employed adults with current asthma, we analyzed 2007–2012 data for 37 505 ever-employed adults (≥18 years) collected from 19 US states (representing an estimated 10 million adults each year). Methods: Using data from landline telephone (LLP) households, we calculated estimates applying poststratification weights (2007–2010) and “raking” weights (2011–2012). Also, using data from LLP/cellular telephone (CP) households combined, we calculated estimates applying “raking” weights (2012). Results: Based on LLP household data, the WRA estimates ranged from 7.8% to 9.7% during 2007–2010, was 9.1% in 2011 and 15.4% in 2012. Possible WRA estimates ranged from 35.1% to 38.1% during 2007–2010, was 38.1% in 2011 and 39.8% in 2012. Using the 2012 LLP/CP household data, the WRA and possible WRA estimates were 15.4% and 38.9%, respectively. Conclusions: Implementation of “raking” weights did not substantially change the WRA or possible WRA estimates among ever-employed adults with current asthma. The WRA and possible WRA estimates based on LLP and LLP/CP samples in 2012 were comparable, as CP users are younger and less likely to have WRA. The substantial upward shift in the 2012 WRA estimates likely was associated with the revision to the WRA section.

Acknowledgements

The authors thank the Behavioral Risk Factor Surveillance System state coordinators for their assistance in collecting the data used in this analysis. The authors also thank Dr Carol Pierannunzi, Division of Population Health, National Center for Chronic Disease Prevention and Health Promotion, Centers for Disease Control and Prevention, Sarah Lyon-Callo, Lifecourse Epidemiology and Genomics Division, Michigan Department of Health and Human Services, and Chris Fussman, Lifecourse Epidemiology and Genomics Division, Michigan Department of Health and Human Services for their thoughtful comments.

Declaration of interest

The authors report no conflicts of interest. This publication was supported by Cooperative Agreement Number 3U36OE000002 from the Centers for Disease Control and Prevention and the Association of Schools and Programs of Public Health. The findings and conclusions in this report are those of the authors and do not necessarily represent the official views of NIOSH, CDC or ASPPH.

Supplementary material available online Supplemental Tables S1–S4

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