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Research Article

A MURINE MODEL OF CIGARETTE SMOKE-INDUCED PULMONARY INFLAMMATION USING INTRANASALLY ADMINISTERED SMOKE-CONDITIONED MEDIUM

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Pages 435-455 | Published online: 02 Jul 2009

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DeepakK. Bhalla, Fusao Hirata, ArunK. Rishi & C. Gary Gairola. (2009) Cigarette Smoke, Inflammation, and Lung Injury: A Mechanistic Perspective. Journal of Toxicology and Environmental Health, Part B 12:1, pages 45-64.
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Kimberly C. Hodge-Bell, K. Monica Lee, Roger A. Renne, Kathy M. Gideon, Sam J. Harbo & Willie J. McKinney. (2007) Pulmonary Inflammation in Mice Exposed to Mainstream Cigarette Smoke. Inhalation Toxicology 19:4, pages 361-376.
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Lisa M. Corey, Coralie Baker & Daniel L. Luchtel. (2006) Heart-Rate Variability in the Apolipoprotein E Knockout Transgenic Mouse Following Exposure to Seattle Particulate Matter. Journal of Toxicology and Environmental Health, Part A 69:10, pages 953-965.
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Margaret K. Elliott, Joseph H. Sisson, William W. West & Todd A. Wyatt. (2006) DIFFERENTIAL IN VIVO EFFECTS OF WHOLE CIGARETTE SMOKE EXPOSURE VERSUS CIGARETTE SMOKE EXTRACT ON MOUSE CILIATED TRACHEAL EPITHELIUM. Experimental Lung Research 32:3-4, pages 99-118.
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K. L. Bailey, J. A. Poole, T. L. Mathisen, T. A. Wyatt, S. G. Von Essen & D. J. Romberger. (2008) Toll-like receptor 2 is upregulated by hog confinement dust in an IL-6-dependent manner in the airway epithelium. American Journal of Physiology-Lung Cellular and Molecular Physiology 294:6, pages L1049-L1054.
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Michael T. Borchers, Scott C. Wesselkamper, Nathaniel L. Harris, Hitesh Deshmukh, Erin Beckman, Mark Vitucci, Jay W. Tichelaar & George D. Leikauf. (2007) CD8+ T cells contribute to macrophage accumulation and airspace enlargement following repeated irritant exposure. Experimental and Molecular Pathology 83:3, pages 301-310.
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Thomas H. Thatcher, Sanjay B. Maggirwar, Carolyn J. Baglole, Heather F. Lakatos, Thomas A. Gasiewicz, Richard P. Phipps & Patricia J. Sime. (2007) Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-κB Component RelB. The American Journal of Pathology 170:3, pages 855-864.
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