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Free Radical Research Volume 34, 2001 - Issue 5
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Original Article

Lipid peroxidation is increased in paraoxonase L55 homozygotes compared with M-allele carriers

Riikka Malin Department of Clinical Chemistry, Tampere University Hospital, Centre for Laboratory Medicine
,
Seppo Laine Medical School, University of Tampere
,
Vappu Rantalaiho Department of Microbiology, Tampere University Hospital
,
Ole Wirta Department of Internal Medicine, P.O. Box 2000, 33521, Tampere
,
Amos Pasternack Department of Mental Health and Alcohol Research, National Public Health Institute, P.O. Box 719, 00101, Helsinki
,
Hannu Jokela Research Unit of Alcohol Diseases, University of Helsinki, Helsinki, Finland
,
Hannu Alho Department of Clinical Chemistry, Tampere University Hospital, Centre for Laboratory Medicine
,
Timo Koivula Medical School, University of Tampere
&
Terho Lehtimäki Department of Microbiology, Tampere University Hospital
 show all
Pages 477-484 | Received 24 Aug 2000, Published online: 07 Jul 2009

Citations (10)

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Wanda Acampa, Maria D. Di Taranto, Alberto Morgante, Barbara Salvatore, Laura Evangelista, Francesca Ricci, Pierluigi Costanzo, Emma de Sisto, Pasquale P. Filardi, Mario Petretta, Giuliana Fortunato & Alberto Cuocolo. (2011) C-reactive protein levels are associated with paraoxonase polymorphism L55M in patients undergoing cardiac SPECT imaging. Scandinavian Journal of Clinical and Laboratory Investigation 71:3, pages 179-184.
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Articles from other publishers (9)

Elyse Di Marco, Jay C. Jha, Arpeeta Sharma, Jennifer L. Wilkinson-Berka, Karin A. Jandeleit-Dahm & Judy B. de Haan. (2015) Are reactive oxygen species still the basis for diabetic complications?. Clinical Science 129:2, pages 199-216.
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Alicia J. Jenkins, Michael A. Hill & Kevin G. Rowley. 2008. Atherosclerosis and Oxidant Stress. Atherosclerosis and Oxidant Stress 123 158 .
Mike J. SampsonSimon BraschiGavin WillisSian B. Astley. (2005) Paraoxonase-1 (PON-1) genotype and activity and in vivo oxidized plasma low-density lipoprotein in Type II diabetes . Clinical Science 109:2, pages 189-197.
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Thomas M. van Himbergen, Lambertus J.H. van Tits, Magda P.C. Hectors, Jacqueline de Graaf, Mark Roest & Anton F.H. Stalenhoef. (2005) Paraoxonase-1 and linoleic acid oxidation in familial hypercholesterolemia. Biochemical and Biophysical Research Communications 333:3, pages 787-793.
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Michael Aviram & Mira Rosenblat. (2004) Paraoxonases 1, 2, and 3, oxidative stress, and macrophage foam cell formation during atherosclerosis development. Free Radical Biology and Medicine 37:9, pages 1304-1316.
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Louise Lyrenäs, Emma Wincent, Lena Forsberg, Ulf de Faire & Ralf Morgenstern. 2003. Redox-Genome Interactions in Health and Disease. Redox-Genome Interactions in Health and Disease.
Jia ChenMadhu KumarWendy ChanGertrud BerkowitzJames G Wetmur. (2003) Increased influence of genetic variation on PON1 activity in neonates.. Environmental Health Perspectives 111:11, pages 1403-1409.
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Cahide Topsakal, Nermin Kilic, Faik Ozveren, Ismail Akdemir, Metin Kaplan, Murat Tiftikci & Ferit Gursu. (2003) Effects of Prostaglandin E1, Melatonin, and Oxytetracycline on Lipid Peroxidation, Antioxidant Defense System, Paraoxonase (PON1) Activities, and Homocysteine Levels in an Animal Model of Spinal Cord Injury. Spine 28:15, pages 1643-1652.
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Giuliana Fortunato, Paolo Rubba, Salvatore Panico, Daniela Trono, Nadia Tinto, Cristina Mazzaccara, Mario De Michele, Arcangelo Iannuzzi, Dino F Vitale, Francesco Salvatore & Lucia Sacchetti. (2003) A paraoxonase gene polymorphism, PON 1 (55), as an independent risk factor for increased carotid intima-media thickness in middle-aged women. Atherosclerosis 167:1, pages 141-148.
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