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Research Article

Evidence of alterations in the expression of orphan receptors GPR26 and GPR39 due to the etiology of the metabolic syndrome

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Pages 422-429 | Received 12 Dec 2016, Accepted 15 Feb 2017, Published online: 07 Mar 2017

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Loranda Calderón-Zamora, Adrian Canizalez-Román, Nidia León-Sicairos, Asdrubal Aguilera-Mendez, Fengyang Huang, Enrique Hong & Santiago Villafaña. (2021) Changes in expression of orphan receptors GPR99 and GPR107 during the development and establishment of hypertension in spontaneously hypertensive rats. Journal of Receptors and Signal Transduction 41:6, pages 558-565.
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Rodrigo Romero-Nava, Noemí García, Karla Aidee Aguayo-Cerón, Fausto Sánchez Muñoz, Fengyang Huang, Enrique Hong & Santiago Villafaña. (2021) Modifications in GPR21 and GPR82 genes expression as a consequence of metabolic syndrome etiology. Journal of Receptors and Signal Transduction 41:1, pages 38-44.
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Articles from other publishers (4)

Rocío Alejandra Gutiérrez-Rojas, Karla Aidee Aguayo-Cerón, Cruz Vargas-De-León, Sandra Edith Cabrera-Becerra, Julio Cesar Almanza-Pérez, Fengyang Huang, Santiago Villafaña & Rodrigo Romero-Nava. (2022) Glycine Effect on the Expression Profile of Orphan Receptors GPR21, GPR26, GPR39, GPR82 and GPR6 in a Model of Inflammation in 3T3-L1 Cells. Life 12:11, pages 1687.
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Zahra Abedi Kichi, Lucia Natarelli, Saeed Sadeghian, Mohammad ali Boroumand, Mehrdad Behmanesh & Christian Weber. (2022) Orphan GPR26 Counteracts Early Phases of Hyperglycemia-Mediated Monocyte Activation and Is Suppressed in Diabetic Patients. Biomedicines 10:7, pages 1736.
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Carmen Alejandrina Virgen-Carrillo, Diana Laura Hernández de los Ríos, Karina Ruíz Torres & Alma Gabriela Martínez Moreno. (2021) Diagnostic Criteria for Metabolic Syndrome in Diet-Induced Rodent Models: A Systematic Review. Current Diabetes Reviews 17:8.
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Irais Rivera-Alvarez, Perla Pérez-Treviño, Héctor Chapoy-Villanueva, Jorge E. Vela-Guajardo, Bianca Nieblas, Salvador Garza-González, Gerardo García-Rivas & Noemí García. (2020) A single session of physical activity restores the mitochondrial organization disrupted by obesity in skeletal muscle fibers. Life Sciences 256, pages 117965.
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