Lipophilic fractions from the marine sponge Halichondria sitiens decrease secretion of pro-inflammatory cytokines by dendritic cells and decrease their ability to induce a Th1 type response by allogeneic CD4⁺ T cells

Xiaxia Dia Faculty of Pharmaceutical Sciences, University of Iceland, Reykjavik, Iceland; ;b Department of Immunology, Landspitali – The National University Hospital of Iceland, Reykjavik, Iceland; View further author information

Jon T. Oskarssonb Department of Immunology, Landspitali – The National University Hospital of Iceland, Reykjavik, Iceland; ;c Centre for Rheumatology Research, Landspitali – The National University Hospital of Iceland, Reykjavik, Iceland; ;d Faculty of Medicine, Biomedical Center, University of Iceland, Reykjavik, IcelandView further author information

Sesselja Omarsdottira Faculty of Pharmaceutical Sciences, University of Iceland, Reykjavik, Iceland; View further author information

Jona Freysdottirb Department of Immunology, Landspitali – The National University Hospital of Iceland, Reykjavik, Iceland; ;c Centre for Rheumatology Research, Landspitali – The National University Hospital of Iceland, Reykjavik, Iceland; ;d Faculty of Medicine, Biomedical Center, University of Iceland, Reykjavik, IcelandView further author information

Ingibjorg Hardardottirb Department of Immunology, Landspitali – The National University Hospital of Iceland, Reykjavik, Iceland; ;d Faculty of Medicine, Biomedical Center, University of Iceland, Reykjavik, IcelandCorrespondence[email protected]
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Lipophilic fractions from the marine sponge Halichondria sitiens decrease secretion of pro-inflammatory cytokines by dendritic cells and decrease their ability to induce a Th1 type response by allogeneic CD4⁺ T cells

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Lipophilic fractions from the marine sponge Halichondria sitiens decrease secretion of pro-inflammatory cytokines by dendritic cells and decrease their ability to induce a Th1 type response by allogeneic CD4+ T cells

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Lipophilic fractions from the marine sponge Halichondria sitiens decrease secretion of pro-inflammatory cytokines by dendritic cells and decrease their ability to induce a Th1 type response by allogeneic CD4⁺ T cells