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Journal of Toxicology and Environmental Health Volume 3, 1977 - Issue 3
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Original Articles

1,1‐Dichloroethylene hepatotoxic1ty: Effect of altered thyroid function and evidence for the subcellular site of injury

Rudolph J. Jaeger Department of Physiology, Harvard School of Public Health, 665 Huntington Avenue, Boston, Massachusetts, 02115
,
Sandor Szabo Department of Pathology, Peter Bent Brigham Hospital, Boston, Massachusetts
&
Linda Jean Coffman Medical School, McGill University, Montreal, Quebec, Canada
Pages 545-555 | Received 24 Jan 1977, Accepted 10 May 1977, Published online: 20 Oct 2009

Citations (13)

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Read on this site (5)

L.A. Videla. (2000) Energy metabolism, thyroid calorigenesis, and oxidative stress: functional and cytotoxic consequences. Redox Report 5:5, pages 265-275.
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X. Boisier, M. Schön, A. Sepúlveda, A. Basualdo, P. Cornejo, C. Bosco, Y. Carrión, M. Galleano, G. Tapia, S. Puntarulo, V. Fernández & L.A. Videla. (1999) Derangement of Kupffer cell functioning and hepatotoxicity in hyperthyroid rats subjected to acute iron overload. Redox Report 4:5, pages 243-250.
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G. Harshani Gunasena & MaryF. Kanz. (1997) EFFECT OF HYPERTHYROIDISM ON THE IN VITRO METABOLISM AND COVALENT BINDING OF 1,1-DICHLOROETHYLENE IN RAT LIVER MICROSOMES. Journal of Toxicology and Environmental Health 52:2, pages 169-188.
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MaryF. Kanz, Lata Kaphalia, Shaheen Mohsin, Zubeda Taj & Mary Treinen Moslen. (1994) Hyperthyroidism increases covalent binding and biliary excretion of 1,1‐dlchloroethylene in rats. Journal of Toxicology and Environmental Health 41:2, pages 187-206.
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D. L. Story, S. J. Gee, C. A. Tyson & D. H. Gould. (1983) Response of isolated hepatocytes to organic and inorganic cytotoxins. Journal of Toxicology and Environmental Health 11:4-6, pages 483-501.
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Articles from other publishers (8)

. 2002. The MAK‐Collection for Occupational Health and Safety. The MAK‐Collection for Occupational Health and Safety 110 139 .
Mary F. Kanz, Zubeda Taj & Mary Treinen Moslen. (1991) 1,1-Dichloroethylene hepatotoxicity: Hypothyroidism decreases metabolism and covalent binding but not injury in the rat. Toxicology 70:2, pages 213-229.
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Mary F. Kanz, Robert F. Whitehead, Ann E. Ferguson & Mary Treinen Moslen. (1988) Potentiation of 1,1-dichloroethylene hepatotoxicity: Comparative effects of hyperthyroidism and fasting. Toxicology and Applied Pharmacology 95:1, pages 93-103.
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Adaline C. Smith, Stephen M. Roberts, Lawrence M. Berman, Raymond D. Harbison & Robert C. James. (1988) Effects of piperonyl butoxide on halothane hepatotoxicity and metabolism in the hyperthyroid rat. Toxicology 50:1, pages 95-105.
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Adaline C. Smith, M.Lawrence Berman, Robert C. James & Raymond D. Harbison. (1983) Characterization of hyperthyroidism enhancement of halothane-induced hepatotoxicity. Biochemical Pharmacology 32:23, pages 3531-3539.
Crossref
Claus-Peter Siegers, Andreas Frühling & Maged Younes. (1983) Halothane hepatotoxicity in hyperthyroid rats as compared to the phenobarbital-hypoxia model. Toxicology and Applied Pharmacology 69:2, pages 257-264.
Crossref
R. J. Jaeger. (1979) Time-related variation of non-protein sulfhydryl concentrations in rat tissues and human blood. International Archives of Occupational and Environmental Health 42:3-4, pages 141-148.
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Rudolph J. Jaeger, Lawrence G. Shoner & Linda Coffman. (1977) 1,1-Dichloroethylene hepatotoxicity: Proposed mechanism of action and distribution and binding of 14 C radioactivity following inhalation exposure in rats . Environmental Health Perspectives 21, pages 113-119.
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