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Original Articles

Developmental toxicity of nine selected compounds following prenatal exposure in the mouse: Naphthalene, p‐nitrophenol, sodium selenite, dimethyl phthalate, ethylenethiourea, and four glycol ether derivatives

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Pages 25-38 | Received 26 Aug 1984, Accepted 11 Nov 1984, Published online: 15 Oct 2009

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Rui Zhao, Nong Xiang, FredrickE. Domann & Weixiong Zhong. (2009) Effects of Selenite and Genistein on G2/M Cell Cycle Arrest and Apoptosis in Human Prostate Cancer Cells. Nutrition and Cancer 61:3, pages 397-407.
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LawrenceA. Kapustka. (2004) Establishing Eco-SSLs for PAHs: Lessons Revealed from a Review of Literature on Exposure and Effects to Terrestrial Receptors. Human and Ecological Risk Assessment: An International Journal 10:2, pages 185-205.
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D. K. Hansen & T. F. Grafton. (1994) Evaluation of di(2‐ethylhexyl)phthalate‐induced embryotoxicity in rodent whole‐embryo culture. Journal of Toxicology and Environmental Health 43:3, pages 361-367.
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DouglasG. Jones & LesleyM. Stevenson. (1993) Influence of Subclinical Excesses of Selenium and/or Vitamin E on Clinical Biochemistry and Antibody Responses in Mice. Journal of Nutritional Immunology 2:1, pages 55-70.
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DennisJ. Paustenbach. (1988) Assessment of the developmental risks resulting from occupational exposure to select glycol ethers within the semiconductor industry. Journal of Toxicology and Environmental Health 23:1, pages 29-75.
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