Adult-onset deactivation of autophagy leads to loss of synapse homeostasis and cognitive impairment, with implications for alzheimer disease

Hilary Grosso Jasutkara Department of Neurology, Columbia University, New York, NY, USA;b Department of Neurology, Rutgers Robert Wood Johnson Medical School, Piscataway, NJ, USACorrespondence[email protected]
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Elizabeth M. Wasserleinc Doctoral Program in Neurobiology and Behavior, Columbia University, New York, NY, USAView further author information

Azeez Isholab Department of Neurology, Rutgers Robert Wood Johnson Medical School, Piscataway, NJ, USAView further author information

Nicole Litta Department of Neurology, Columbia University, New York, NY, USAView further author information

Agnieszka Staniszewskid The Taub Institute for Research on Alzheimer’s Disease and the Aging Brain, Columbia University, New York, NY, USAView further author information

Ottavio Aranciod The Taub Institute for Research on Alzheimer’s Disease and the Aging Brain, Columbia University, New York, NY, USA;e Department of Pathology and Cell Biology, Columbia University, New York, NY, USAView further author information

Ai Yamamotoa Department of Neurology, Columbia University, New York, NY, USA;e Department of Pathology and Cell Biology, Columbia University, New York, NY, USACorrespondence[email protected]
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Adult-onset deactivation of autophagy leads to loss of synapse homeostasis and cognitive impairment, with implications for alzheimer disease

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Adult-onset deactivation of autophagy leads to loss of synapse homeostasis and cognitive impairment, with implications for alzheimer disease

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