Lipopolysaccharide-Induced NF-κB Activation in Mouse 70Z/3 Pre-B Lymphocytes Is Inhibited by Mevinolin and 5′-Methylthioadenosine: Roles of Protein Isoprenylation and Carboxyl Methylation Reactions

Ronald E. Law1 Departments of Microbiology and Immunology, University of California, Los Angeles, California90024;2 Molecular Biology Institute, University of California, Los Angeles, California90024

Julie B. Stimmel2 Molecular Biology Institute, University of California, Los Angeles, California90024;3 Chemistry and Biochemistry, University of California, Los Angeles, California90024

Michael A. Damore2 Molecular Biology Institute, University of California, Los Angeles, California90024

Carla Carter1 Departments of Microbiology and Immunology, University of California, Los Angeles, California90024;2 Molecular Biology Institute, University of California, Los Angeles, California90024

Steven Clarke2 Molecular Biology Institute, University of California, Los Angeles, California90024;3 Chemistry and Biochemistry, University of California, Los Angeles, California90024

Randolph Wall1 Departments of Microbiology and Immunology, University of California, Los Angeles, California90024;2 Molecular Biology Institute, University of California, Los Angeles, California90024

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Mei-Ling Yang, Alaric J.P. Gee, Renelle J. Gee, Cecilia I. Zurita-Lopez, Shilpi Khare, Steven G. Clarke & Mark J. Mamula. (2013) Lupus autoimmunity altered by cellular methylation metabolism. Autoimmunity 46:1, pages 21-31.
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Lipopolysaccharide-Induced NF-κB Activation in Mouse 70Z/3 Pre-B Lymphocytes Is Inhibited by Mevinolin and 5′-Methylthioadenosine: Roles of Protein Isoprenylation and Carboxyl Methylation Reactions

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Lipopolysaccharide-Induced NF-κB Activation in Mouse 70Z/3 Pre-B Lymphocytes Is Inhibited by Mevinolin and 5′-Methylthioadenosine: Roles of Protein Isoprenylation and Carboxyl Methylation Reactions

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